ORCID Profile
0000-0002-3032-6410
Current Organisations
Royal Veterinary College
,
Hashemite University
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Publisher: Public Library of Science (PLoS)
Date: 21-06-2016
Publisher: DJ Studio Dariusz Jasinski
Date: 30-04-2022
DOI: 10.56984/8ZG0DE8MY
Abstract: Introduction. Coronavirus disease 2019 (COVID-19) is an emerging pandemic disease caused by the severe acute respiratory syndrome. Back pain could be triggered by the COVID-19 virus either directly or indirectly. However, to our knowledge, limited studies are addressing the development of chronic back pain or new-onset of back pain among COVID-19 survivors. Therefore, the purpose of this study was to investigate the potential health consequences of COVID-19 on back pain among the COVID-19 survivors. Methods. We used an electronically distributed survey targeting adult Jordanian citizens and residents who recovered from COVID-19 virus infection. The questionnaire composed questions regarding demographic characteristics, COVID-19 and spine injury-related information. Results. 27.2% of participants reported increase of back pain with COVID during the episode and 16% of participants reported increased back pain after COVID with pain lasted less than 3 months in 16.6% and more than 3 months in 17.8% of them. There was significant increase on back pain level during COVID compared to pre COVID. At the same time there was significant increase on back pain post COVID compared to pre COVID. Conclusion. The COVID-19 virus resulted in a significant increase in spine pain among COVID-19 survivors and it lead to chronic back pain among patients with risk factors and some develop new spine pain.
Publisher: Ovid Technologies (Wolters Kluwer Health)
Date: 19-02-2020
DOI: 10.1097/AJP.0000000000000822
Abstract: Conditioned pain modulation (CPM) and manipulation-induced analgesia (MIA) are 2 forms of endogenous analgesia. Many forms of analgesia can be influenced by the nature of the patient-clinician interaction. The aim of this study was to evaluate the influence of an empathetic and supportive interaction on CPM and MIA in people with lateral epicondylalgia (LE). In a double-blind, randomized, controlled trial, 68 participants with LE were assigned to 2 groups: the empathetic and neutral interaction groups. The interactions were carried out by a trained, professional role-play actor, playing the part of a research assistant. The research assistant actor spent 15 minutes before CPM and MIA assessment interacting with the participants in an empathetic or neutral manner. Immediately after the interaction, a blinded assessor measured pressure pain threshold at the symptomatic elbow and ipsilateral wrist during CPM and MIA testing. Linear mixed models were used to evaluate differences in CPM and MIA responses between the interaction groups. There was a significant difference in Consultation and Relational Empathy scores between the groups ( P .001), indicating that the intervention group experienced a more empathic interaction. Both groups showed a significant increase in pressure pain threshold measures, indicative of a CPM and MIA analgesic response ( P .001), however, the analgesic responses were greater in the group that had experienced a supportive, empathetic interaction (post CPM, wrist: P .001 elbow: P =0.001) (post MIA wrist: P .001 elbow: P =0.001). A single session of empathetic interaction positively influenced both CPM and MIA responses in people with LE.
Publisher: Research Square Platform LLC
Date: 11-08-2020
DOI: 10.21203/RS.3.RS-42581/V1
Abstract: Background: Exercise has been shown to reduce pain sensitivity. It is unclear whether exercise can also potentiate the endogenous analgesia and reduction in pain sensitivity produced by conditioned pain modulation (CPM) and manipulation induced analgesia (MIA). Objectives: To determine whether aerobic exercise potentiates CPM and MIA and to investigate associations between exercise induced analgesia, CPM and MIA. Design: Parallel randomised, controlled trial using computer-generated randomisation to allocate interventions. Methods: A gender-stratified convenience s le included 68 participants with lateral epicondylalgia (LE) from Perth, Western Australia, recruited between October 2017 and June 2018. Participants were allocated to receive either moderate intensity aerobic exercise (control condition, n=34) or high intensity aerobic exercise (active condition, n=34) for 15 minutes on two separate test sessions, with a three-day rest in between. Exercise intensity was determined based on age-related target heart rate (HR) corresponding to 50% and 75% of maximum HR respectively. Following aerobic exercise, participants were immediately assessed for CPM or MIA response, one on each test day, in a random order. A blinded assessor measured pressure pain thresholds (PPT), the main outcome measure, at the elbow and ipsilateral wrist to evaluate CPM and MIA. Data were analysed using linear mixed models, partial correlations, and univariate regression. Results: All participants showed significant increases in PPT at both test sites (p .001). The high intensity exercise group demonstrated higher levels of CPM and MIA at both test sites (p .001). There were large and positive partial correlations between CPM/MIA and the initial change in PPT following exercise (CPM (r:0.90–0.93, p .001)/MIA (r:0.68–0.86, p .001)). The change in PPT following aerobic exercise was a significant predictor of both CPM (adjusted R 2 :92%-95%) and MIA (adjusted R 2 :73%-93%) response. Conclusion: An acute bout of high intensity aerobic exercise significantly enhanced the analgesic effect of CPM and MIA in people with LE. CPM and MIA may activate similar descending inhibitory mechanisms to mediate their analgesic effects. Trial registration : Prospectively registered with the Australia New Zealand Clinical Trials Registry on 9/02/2017: ACTRN12617000219381, anzctr.org.au/Trial/Registration/TrialReview.aspx?id=372232
Publisher: Ovid Technologies (Wolters Kluwer Health)
Date: 05-2019
DOI: 10.1097/AJP.0000000000000696
Abstract: Conditioned pain modulation (CPM) and manipulation-induced analgesia (MIA) may activate similar neurophysiological mechanisms to mediate their analgesic effects. This study assessed the association between CPM and MIA responses in people with lateral epicondylalgia. Seventy participants with lateral epicondylalgia were assessed for CPM followed by MIA. A single assessor measured pressure pain thresholds (PPT) before, during, and after cold water immersion (10°C) of the asymptomatic hand and contralateral lateral glide (CLG) mobilization of the neck. For analyses, linear mixed models evaluated differences in CPM and MIA responses. Pearson partial correlations and regression analyses evaluated the association between CPM and MIA PPT. There was a significant increase (CPM and MIA, P .001) in PPT from baseline during the interventions (CPM mean: 195.84 kPa for elbow and 201.87 kPa for wrist, MIA mean: 123.01 kPa for elbow and 126.06 kPa for wrist) and after the interventions (CPM mean: 126.06 kPa for elbow, 114.24 kPa for wrist, MIA mean: 123.50 kPa for elbow and 122.16 kPa for wrist). There were also significant moderate and positive partial linear correlations ( r : 0.40 to 0.54, P .001) between CPM and MIA measures, controlling for baseline measures. Regression analyses showed that CPM PPT was a significant predictor of MIA PPT ( P .001) and the models explained between 73% and 85% of the variance in MIA PPT. This study showed that CPM and MIA responses were significantly correlated and that the CPM response was a significant predictor of MIA response.
Location: United Kingdom of Great Britain and Northern Ireland
Location: United Kingdom of Great Britain and Northern Ireland
Location: United Kingdom of Great Britain and Northern Ireland
Location: United Kingdom of Great Britain and Northern Ireland
No related grants have been discovered for Ahmad Muhsen.