Characterisation of the pathogenesis of FHL1 myopathies

Funding Activity

Website
http://purl.org/au-research/grants/nhmrc/GNT1104615

Funding Status
Closed

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Funded Activity Summary

Skeletal muscle is the most abundant tissue in the body and dynamically capable of responding to many environmental stresses. A key cellular process that has developed in muscle to facilitate adaptive responses is autophagy, a mechanism that facilitates the degradation and recycling of cellular debris. Defects in autophagy cause muscle disease. In this study we will identify a novel gene that regulates autophagy and will investigate how mutations in this gene cause muscle disease.

Funded Activity Details

Start Date: 01-01-2016

End Date: 01-01-2019

Funding Scheme: Project Grants

Funding Amount: $748,652.00

Funder: National Health and Medical Research Council

Research Topics

ANZSRC Field of Research (FoR)

Biochemistry and Cell Biology not elsewhere classified

ANZSRC Socio-Economic Objective (SEO)

There are no SEO codes available for this funding activity

Other Keywords

autophagy | mouse models | muscular dystrophy | protein aggregation | skeletal muscle

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