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Field of Research : Medical Virology
Research Topic : virus vector
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  • Funded Activity

    Wolbachia And West Nile Virus In Mosquitoes: Friends Or Foes?

    Funder
    National Health and Medical Research Council
    Funding Amount
    $561,028.00
    Summary
    Mosquito-borne viruses pose a great risk to human and animal health. Presence of compentent vectors of several viruses in Australia indicates vulnerability of Australia’s biosecurity. This project will define the mechanisms of inhibition of virus replication in mosquitoes by a symbiotic bacterium which can be utilized in virus inhibition.
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    Funded Activity

    Role Of Flavivirus-encoded Small Regulatory RNAs In Virus-mosquito Vector Interactions

    Funder
    National Health and Medical Research Council
    Funding Amount
    $547,216.00
    Summary
    Mosquito-borne diseases are major threats to human health. MicroRNAs are small non-coding ribonucleic acids (RNAs) that play important roles in development, cancer, apoptosis, immunity, longevity, and viral infections. We propose to identify the regulatory microRNAs from flaviviruses and establish their potential function in vector-arboviruses interactions. The project will put Australia at the forefront of research in the most rapidly developing area of microRNA research.
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    Funded Activity

    Dengue Pathogenesis And Disease Control

    Funder
    National Health and Medical Research Council
    Funding Amount
    $652,765.00
    Summary
    Dengue is a serious viral disease that is spread by mosquitoes. Dengue results in hundreds of thousands of children and young adults being hospitalized in Asia every year, and some patients die. There is no vaccine or specific treatment. In this fellowship I will lead a research program that aims provide doctors with the tools to diagnosis and treat dengue more efficiently and to develop a novel biological control to prevent dengue being transmitted by mosquitoes.
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    Funded Activity

    Resolving Human Immunodeficiency Virus (HIV) Transmission

    Funder
    National Health and Medical Research Council
    Funding Amount
    $745,213.00
    Summary
    To increase the breadth of HIV prevention strategies, it is imperative that we biologically understand how HIV enters our bodies. Through two unique clinical cohorts, we will determine why circumcision is protective and how a commonly acquired sexual transmitted infection (human papilloma virus) can increase HIV transmission.
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    Funded Activity

    Intrinsic Host Antiviral Activity Against Pathogenic Filoviruses

    Funder
    National Health and Medical Research Council
    Funding Amount
    $488,754.00
    Summary
    Bats are a major reservoir for deadly human viruses including Ebola and Marburg virus. In contrast to humans, bats can be infected with these viruses without showing clinical signs of disease. The reason why bats can co-exist with these viruses is unknown. This study will determine if a bat antiviral molecule contributes to limiting virus release compared to the human version that could reveal strategies to prevent and control these deadly viruses in humans.
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    Funded Activity

    Identification Of Host Factors That Restrict Influenza Virus Replication In Macrophages

    Funder
    National Health and Medical Research Council
    Funding Amount
    $566,446.00
    Summary
    Influenza virus infects different cells in the airways, including immune cells (macrophages) and non-immune cells (epithelial cells). Epithelial cell infection results in virus amplification and release whereas macrophage infection leads to virus destruction. This project will identify cellular factors expressed by macrophages that block virus amplification and release. Identification of novel antiviral factors is an important step towards developing strategies to reduce influenza disease.
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    Funded Activity

    Identification Of Host Restriction Factors That Block Respiratory Virus Infection

    Funder
    National Health and Medical Research Council
    Funding Amount
    $956,898.00
    Summary
    Following inhalation, respiratory viruses can infect and grow in airway epithelial cells. Although immune cells such as macrophages are also susceptible to infection, this is generally abortive and new viruses are not released. This project will identify proteins induced in macrophages that block respiratory viruses and prevent their spread in the airways. We will also define mechanisms by which some virulent strains overcome this block to grow in macrophages.
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    Funded Activity

    The Role Of Varicella Zoster Virus In Modulating Cutaneous Infection

    Funder
    National Health and Medical Research Council
    Funding Amount
    $555,892.00
    Summary
    Varicella zoster virus (VZV) causes two skin diseases: chickenpox and shingles. VZV can causes significant morbidity in children and adults and life-threatening disease in immunocompromised people. This project aims to improve our understanding of how VZV affects the function of specialised skin cells to provide information for the development of a better vaccine to lessen the impact of VZV disease on the community.
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    Funded Activity

    Comparative Expression Studies To Identify Cellular Factors Promoting Hendra Virus Replication For A Comprehensive Understanding Of Hendra Virus Pathogenesis

    Funder
    National Health and Medical Research Council
    Funding Amount
    $374,619.00
    Summary
    Hendra virus (HeV) is an emerging pathogen indigenous to fruit bats. HeV is associated with limited outbreaks with high mortality in domesticated animals and humans. To advance the understanding of HeV-related pathogenesis, we will perform comparative studies in bat and human cell lines to recognise differences in virus-host cell interactions leading to a comprehensive understanding of the HeV life cycle and pathogenesis.
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    Funded Activity

    Structure And Function Of The HCV Glycoproteins

    Funder
    National Health and Medical Research Council
    Funding Amount
    $598,863.00
    Summary
    Hepatitis C Virus infects 3% of the world's population causing recurring liver disease, cirrhosis and hepatocellular carcinoma. To infect a liver cell, the viral glycoproteins attach to cell surface molecules wher they are activated to mediate merger of the viral and cellular membranes. This project grant will explore how the viral glycopropteins become activated and obtain essential structural information on the viral glycoproteins. These studies will help us to design antiviral agents.
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