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Research Topic : knockout mouse
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  • Funded Activity

    Vitamin D Synthesis Within Osteoblasts Increases Bone Mineral By Regulating Remodelling: Is This The Link Between Vitamin D Status And Fractures?

    Funder
    National Health and Medical Research Council
    Funding Amount
    $627,082.00
    Summary
    This project will contribute to understanding mechanism of vitamin D action within bone to modulate bone resorption and offers the exciting prospect of identifying the mechanism by which an adequate vitamin D status can reduce the risk of osteoporotic hip fractures. Thus, this project has great potential to improve community health by being able to recommend vitamin D supplementation made on the basis of maintaining normal bone cell function with psarticular reference to modulating bone resorpti .... This project will contribute to understanding mechanism of vitamin D action within bone to modulate bone resorption and offers the exciting prospect of identifying the mechanism by which an adequate vitamin D status can reduce the risk of osteoporotic hip fractures. Thus, this project has great potential to improve community health by being able to recommend vitamin D supplementation made on the basis of maintaining normal bone cell function with psarticular reference to modulating bone resorption.
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    Funded Activity

    Elucidating The Role Of MiR-196 In Formation Of The Axial Skeleton

    Funder
    National Health and Medical Research Council
    Funding Amount
    $520,087.00
    Summary
    Exquisite regulation of gene expression is a fundamental principle underlying growth and development of an embryo as well as homeostasis in the adult. Following the identification of hundreds of microRNAs within the genome which act to modulate gene expression, the challenge and the goal of these studies, is to identify individual microRNAs which contribute significantly to bone formation in the developing embryo.
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    Funded Activity

    Molecular Characterization Of V-ATPase V0 Domain Subunits E1 And E2 In Osteoclast

    Funder
    National Health and Medical Research Council
    Funding Amount
    $558,909.00
    Summary
    Osteoporotic fractures in the elderly are often linked to increased mortality rates. Excess bone resorption is a major contributor to the onset of the disease. The proposed project focuses on the investigation of the molecular mechanisms of acid secretion that is required for the bone degradation in body. The project will examine the role of the proton pump in bone resorption and seek potential targets for the treatment of osteoporosis.
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    Funded Activity

    Epigenetic Regulation Of L1 Retrotransposition In Mouse Models Of Abnormal Human Neurobiology

    Funder
    National Health and Medical Research Council
    Funding Amount
    $417,812.00
    Summary
    Retrotransposons are mobile genes that copy-and-paste themselves to spread in DNA. Until very recently, they were thought to only be active in sperm and egg. In our recent work, we demonstrated that they also move in the brain. In the current study, we will use cutting-edge technologies to determine how retrotransposons change the genetic makeup of neurons in neurodevelopmentally impaired mice to predict whether these mutations would also be present in human brain disorders.
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    Funded Activity

    Deciphering The Overlapping Roles Of SSB1 And SSB2 In The Regulation Of Haematopoiesis And Intestinal Homeostasis

    Funder
    National Health and Medical Research Council
    Funding Amount
    $996,631.00
    Summary
    Our work centres on elucidating the role of two newly identified and related single-stranded DNA binding protein (Ssb1 and Ssb2) in development of blood and gut system. When both genes are deleted mice die with 8 days of knockdown due to bone marrow failure and intestinal atrophy. Our double knockout model parallels the consequences of radiation damage on blood and gut system. Toxicity to these systems is a significant hindrance in delivering anti-tumor therapy.
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    Funded Activity

    DISSECTING THE ROLE OF NEUROLIGINS IN COGNITION

    Funder
    National Health and Medical Research Council
    Funding Amount
    $526,767.00
    Summary
    Problems in learning, memory and other complex mental processes are common to many brain disorders. This project will study the impact of mutations on a family of genes reported in autism and schizophrenia, on complex cognitive behaviours using novel behavioural technologies. This will not only shed fundamental insights into the specific mental processes regulated by these genes and their role in disease, but importantly provide novel targets for the development of therapies.
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    Funded Activity

    Identification Of Genes Causing Medulloblastoma By Transposon Mutagenesis.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $621,997.00
    Summary
    Brain tumours are the most common cause of cancer-related death in children and the tumour medulloblastoma is the most frequent. There is a need to develop new therapeutic approaches to treating medulloblastoma through the development of new drugs to directly target the tumour. This research has identified new genes that are good candidates as drug targets for treating medulloblastoma.
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    Funded Activity

    Metabolic Control Of Innate-like Lymphocytes

    Funder
    National Health and Medical Research Council
    Funding Amount
    $626,563.00
    Summary
    All cells in the body need to get their energy from somewhere, and the chemical basis of their energy supply varies depends on many factors, including their location and rate of cell division. We have found that an important population of white blood cells that control the character and magnitude of most immune responses appear to use an unusual source of their energy. If true this would provide a range of new opportunities to control the numbers and activities of these cells, a thereby control .... All cells in the body need to get their energy from somewhere, and the chemical basis of their energy supply varies depends on many factors, including their location and rate of cell division. We have found that an important population of white blood cells that control the character and magnitude of most immune responses appear to use an unusual source of their energy. If true this would provide a range of new opportunities to control the numbers and activities of these cells, a thereby control the character and magnitude of immune responses.
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    Funded Activity

    Defining Ubiquitin Ligase Substrates: New Therapeutic Strategies In Breast Cancer

    Funder
    National Health and Medical Research Council
    Funding Amount
    $598,163.00
    Summary
    Current cancer therapies use drugs that target both tumor cells and rapidly growing normal cells – causing side effects and limiting effectiveness. Newer treatments aim to target molecules that are unique to tumor cells, leaving normal cells unharmed. This project will study a process that tags proteins for destruction by a cellular recycling system, which is often disrupted in cancer. This research will not only help us understand how cancer develops, but also identify new targets for therapy.
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    Funded Activity

    The Role Of Heg-CCM2L Signaling In Angiogenesis

    Funder
    National Health and Medical Research Council
    Funding Amount
    $665,457.00
    Summary
    Dysfunctional blood vessel growth is an important mechanism of many congenital vascular diseases and other postnatal diseases such as ischemia and cancer. Cerebral cavernous malformations (CCMs) are common vascular disease in brain that cause strokes and seizures in midlife. Due to their location in the brain, CCMs are virtually untreatable, making the development of novel therapies a priority. This proposal aims to understand how the molecular players underlying this brain vascular disease cont .... Dysfunctional blood vessel growth is an important mechanism of many congenital vascular diseases and other postnatal diseases such as ischemia and cancer. Cerebral cavernous malformations (CCMs) are common vascular disease in brain that cause strokes and seizures in midlife. Due to their location in the brain, CCMs are virtually untreatable, making the development of novel therapies a priority. This proposal aims to understand how the molecular players underlying this brain vascular disease control blood vessel function and growth.
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