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Research Topic : interleukin-21
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  • Researchers (0)
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  • Funded Activity

    The Role Of Interleukin-21 In The Pathogenesis Of Autoimmune Diabetes

    Funder
    National Health and Medical Research Council
    Funding Amount
    $489,060.00
    Summary
    Interleukin-21 (IL-21) is a soluble protein that is produced by cells enabling them to communicate with other cells. IL-21 helps cells to clear viruses and bacteria from the body. However, our studies show that IL-21 also generates T cells that destroy beta cells and cause diabetes. IL-21 is produced at abnormally high levels in an important murine model of spontaneous type-1 diabetes (T1D) and if we block IL-21 we prevent diabetes. This projects' aims assess IL-21 as therapeutic target for T1D.
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    Funded Activity

    Mechanisms Underlying The Upregulation Of Cytokine Genes In Allergy

    Funder
    National Health and Medical Research Council
    Funding Amount
    $670,826.00
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    Funded Activity

    The Role Of Interleukin-21 In The Pathogenesis Of Autoimmune Diabetes

    Funder
    National Health and Medical Research Council
    Funding Amount
    $519,000.00
    Summary
    T cells are a component of our blood (white blood cells) and a major component of the body's defense system against infection, known as immunity. Without T cells, we would fail to resist infection by foreign agents, such as viruses, bacteria and fungi. Autoimmune (type 1) diabetes is a disease in which T cells attack our own pancreatic islet self tissues as if they were foreign. T cells that react against the islets of the pancreas cause destruction of the insulin producing beta cells so that th .... T cells are a component of our blood (white blood cells) and a major component of the body's defense system against infection, known as immunity. Without T cells, we would fail to resist infection by foreign agents, such as viruses, bacteria and fungi. Autoimmune (type 1) diabetes is a disease in which T cells attack our own pancreatic islet self tissues as if they were foreign. T cells that react against the islets of the pancreas cause destruction of the insulin producing beta cells so that the pancreas can no longer make insulin. Diabetes is a life-threatening disease because insulin is a hormone that enables people to get energy from food. Type 1 diabetes is usually diagnosed in childhood and insulin must be administered daily by injection or through a pump in order to survive. Unfortunately, taking insulin doesn t cure diabetes and people continue to suffer from an extensive list of complications affecting most vital organs. Interleukin-21 (IL-21) is a soluble protein that is produced by cells enabling them to communicate with other cells. IL-21 helps cells to produce factors that cause inflammation and assist in clearance of viruses and bacteria from the body. However, our studies show that IL-21 is a major factor in the development of the T cells that destroy beta cells and cause diabetes. Our studies show that IL-21 is over-expressed in an important murine model of spontaneous type-1 diabetes. We have isolated the T cells that cause diabetes and show that they are distinguished from other T cells by very high levels of the receptor for IL-21. This project focuses on the IL-21-responsive T cells that cause diabetes and aims to determine the mechanisms by which the cytokine IL-21 causes destructive immune responses and ways to modulate its production. This project applies basic science to the important public health issue of type 1 diabetes for the development of therapeutic intervention strategies.
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    Funded Activity

    Precision Molecular Diagnostics Of Single Cells By Imaging Flow Cytometry

    Funder
    National Health and Medical Research Council
    Funding Amount
    $875,110.00
    Summary
    We have invented a highly sensitive, new method to count and evaluate chromosomes inside cancer cells. With this world-first method we can detect abnormal chromosomes when fewer than 1 cell in 1,000 is affected. We will refine the method for assessment of blood cancers and chromosome disorders such as trisomy 21 (Down Syndrome), and commence pre-clinical testing. This is a critical step towards translating this methodology into a validated test to significantly benefit human health.
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    Funded Activity

    Dysregulation Of The Interleukin-7/Interleukin-7 Receptor Axis In HIV-1 Infection

    Funder
    National Health and Medical Research Council
    Funding Amount
    $43,319.00
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    Funded Activity

    Characterisation Of The Signalling Pathways Utilised By IL-13 Independently Of IL-4R Chain To Mediate Aspects Of Allerg

    Funder
    National Health and Medical Research Council
    Funding Amount
    $259,015.00
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    Funded Activity

    The Biological Function Of Genes On Human Chromosome 21 And Their Role In The Pathophysiology Of Down Syndrome

    Funder
    National Health and Medical Research Council
    Funding Amount
    $2,821,602.00
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    Funded Activity

    Molecular Mechanisms In The Development Of Asthma

    Funder
    National Health and Medical Research Council
    Funding Amount
    $304,770.00
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    Funded Activity

    Ets2 And Erg Genes In Down Syndrome

    Funder
    National Health and Medical Research Council
    Funding Amount
    $191,999.00
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    Funded Activity

    The Mechanism By Which Lymphocytes Are Turned On And St Udies Of A Disease In Which The Mechanism Is Blocked

    Funder
    National Health and Medical Research Council
    Funding Amount
    $284,139.00
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