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Field of Research : Clinical chemistry (incl. diagnostics)
Research Topic : antigen receptor
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  • Funded Activity

    The Structural Basis Of T-cell Allorecognition

    Funder
    National Health and Medical Research Council
    Funding Amount
    $36,611.00
    More information
    Funded Activity

    Exploring The Contribution Of The Immunoproteasome To Immunodominance And T Cell Function

    Funder
    National Health and Medical Research Council
    Funding Amount
    $82,421.00
    Summary
    The immunoproteasome is a piece of cellular machinery which degrades proteins and has been shown to enhance the body's recognition and response to viruses and cancer cells. This immunoproteasome is made up of various subunits, but it has not yet been assessed how each of these subunits contribute to the overall response. By studying the individual subunits, we will have a better understanding in how to manipulate the immune system for anti-viral and anti-cancer vaccine design.
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    Funded Activity

    Tracking Endogenous Presentation Of MHC Class-II-Restricted Viral Epitopes

    Funder
    National Health and Medical Research Council
    Funding Amount
    $165,436.00
    Summary
    CD4+ T cells play an important role in controlling viral infections. Proteins from viruses are processed into small pieces by immune stimulating cells and these are then displayed on special molecules of the immune stimulating cells for the CD4+ T cells to recognise and respond to. This project aims to establish the various pathways by which the immune stimulating cells process the proteins and present them to the CD4+ T cells.
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    Funded Activity

    Functional Suicide Of Selected Dendritic Cells By Cytochrome C: An In Vivo Model Lacking Cross-presentation

    Funder
    National Health and Medical Research Council
    Funding Amount
    $597,476.00
    Summary
    Certain white blood cells (dendritic cells) activate the immune system, especially its T cells. Infection of such cells elicits killer T cell responses. However not all infections infect dendritic cells. In such cases, the infectious material is eaten by dendritic cells and moved to certain areas within the cell. This process is called cross-presentation and how important it is during various diseases remains moot. We now have a model of testing this by eliminating these cross-presenting cells.
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    Funded Activity

    The Genetics Controlling The Course Of Herpesvirus Infection In Humans

    Funder
    National Health and Medical Research Council
    Funding Amount
    $69,684.00
    Summary
    Epstein-Barr Virus and Human Cytomegalovirus are common viruses present in more than 50% of the adult population. They have a major impact on the human immune system, stimulating large numbers of T cells that are essential to control the persistent viral infection. Generally, these viruses cause few problems; however, an unfortunate minority suffer major life threatening clinical problems. There has also been some evidence for a role of EBV infection in the pathogenesis of Multiple Sclerosis.
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    Funded Activity

    Antigen Presentatiion, Recognition And The Immune Response

    Funder
    National Health and Medical Research Council
    Funding Amount
    $7,822,981.00
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    Funded Activity

    Mechanisms Regulating Antigen Presentation During Primary And Recall Responses Of T Cells Following Pathogen Infection

    Funder
    National Health and Medical Research Council
    Funding Amount
    $441,364.00
    Summary
    The primary role of the immune system is the containment of pathogens, cancer cells and infections. This depends on generating long-lived memory CD8+ killer T lymphocytes. Generally this process is achieved efficiently in acute infections in which the pathogen grows relatively rapidly. However, pathogens such as herpes viruses and tuberculosis grow more slowly, fail to efficiently activate the killer T cells such that they elude the immune system and are never completely removed from the body. T .... The primary role of the immune system is the containment of pathogens, cancer cells and infections. This depends on generating long-lived memory CD8+ killer T lymphocytes. Generally this process is achieved efficiently in acute infections in which the pathogen grows relatively rapidly. However, pathogens such as herpes viruses and tuberculosis grow more slowly, fail to efficiently activate the killer T cells such that they elude the immune system and are never completely removed from the body. These latter infections result in persistent or chronic infections. Our work will endeavour to unravel the mechanisms that underlie how a killer T cell is effectively activated and the factors that contribute to failure of these cells to be similarly activated in a persistent infection. The central aim of the studies described in this proposal is to understand the mechanisms utilized by different pathogens to generate the diverse population of memory killer T cells that allow us to respond to the plethora of pathogens we might encounter every day. These studies will improve our understanding of how antigen presenting cells and killer T lymphocytes ensure an immune response is maintained and may identify checkpoints that could be targeted to modulate the immune response when it goes wrong.
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    Funded Activity

    The Role Of TRAF2 Signal Transduction Molecule In Lymphocyte Responses

    Funder
    National Health and Medical Research Council
    Funding Amount
    $90,288.00
    More information
    Funded Activity

    The Role Of Antigen Presentation In The CD8+T Cell Responseto Herpes Simplex Virus-1

    Funder
    National Health and Medical Research Council
    Funding Amount
    $167,146.00
    More information
    Funded Activity

    The Role Of The P2x7 Purinergic Receptors In The Control Of Tuberculosis Infection By Macrophages

    Funder
    National Health and Medical Research Council
    Funding Amount
    $100,721.00
    More information

    Showing 1-10 of 24 Funded Activites

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