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Research Topic : adrenergic
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  • Funded Activity

    Specificity Of Alpha1-Adrenergic Receptor Signalling In Cardiac Hypertrophy And Ischaemia.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $217,093.00
    More information
    Funded Activity

    Effects Of Hypoxia & Ischemia On Sensitivity Of Ion Channels To Beta&alpha-adrenergic Recept In Card Myocytes

    Funder
    National Health and Medical Research Council
    Funding Amount
    $239,109.00
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    Funded Activity

    Development Of Small Molecule Adrenergic Ligands For Single Effector Pathway Activation

    Funder
    National Health and Medical Research Council
    Funding Amount
    $162,116.00
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    Funded Activity

    Mechanisms Of Hormone And Drug Action

    Funder
    National Health and Medical Research Council
    Funding Amount
    $415,120.00
    More information
    Funded Activity

    Electrical Events In Secreting Glands

    Funder
    National Health and Medical Research Council
    Funding Amount
    $92,911.00
    More information
    Funded Activity

    Stress And Diabetes: Effects Of Adrenaline On How Body Tissues Handle Glucose

    Funder
    National Health and Medical Research Council
    Funding Amount
    $157,052.00
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    Funded Activity

    Integrated Drug Design For A New Generation Of Adrenergic Therapeutics

    Funder
    National Health and Medical Research Council
    Funding Amount
    $406,242.00
    Summary
    Fundamental to our ability to respond to both immediate and long-term environmental changes and stresses is the coordinated regulation of cellular functions by hormonal and neurotransmitter stimuli. The great majority of such stimuli are sensed by complex glycoprotein receptors on the surface of most cells that selectively bind and are activated by various hormones and neurotransmitters. Although there are several hundred distinct, but structurally related receptors of this kind, including the a .... Fundamental to our ability to respond to both immediate and long-term environmental changes and stresses is the coordinated regulation of cellular functions by hormonal and neurotransmitter stimuli. The great majority of such stimuli are sensed by complex glycoprotein receptors on the surface of most cells that selectively bind and are activated by various hormones and neurotransmitters. Although there are several hundred distinct, but structurally related receptors of this kind, including the adrenergic receptors (ARs), the molecular mechanisms involved in their activation and, thus, their regulation of vital cellular functions, remain unclear. Based on insights that we have gained from the development and characterisation of several mutated ARs, we have developed a model of receptor activation. In this application we propose to further test and extend the hypotheses underlying this model. Importantly, the functions regulated by ARs include vital responses, such as the maintenance of blood pressure by augmenting heart pump function and by constricting vascular smooth muscle. In addition, disordered cellular regulation by ARs has been implicated in a wide variety of diseases, including high blood pressure, congestive heart failure and enlargement of the heart. Thus, the studies detailed here to further understand the molecular mechanisms of receptor activation have broad implications for our knowledge of critical physiological control systems, and may lead to novel therapeutic approaches to treat a variety of diseases, including also tumours of the adrenal gland that cause excess adrenaline secretion. The cost and length of time associated with the development of a new drug in the pharmaceutical industry are enormous, and thus many promising medicinal agents never make it to the market. We propose to contribute to the drug discovery effort by developing novel combined methods for computer-aided drug design, to allow more efficient drug development.
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    Funded Activity

    The Control Of Protein Secretion In Exocrine Glands

    Funder
    National Health and Medical Research Council
    Funding Amount
    $129,271.00
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    Funded Activity

    Alteration Of Glucose Metabolism By GPCR Activation

    Funder
    National Health and Medical Research Council
    Funding Amount
    $444,796.00
    Summary
    In type 2 diabetes the effect of insulin to stimulate glucose transport in fat cells and skeletal muscle is impaired so there is great interest in identifying insulin-independent mechanisms that increase glucose transport. Several G protein-coupled receptors (GPCRs) regulate glucose transport independently of insulin but the mechanisms involved in these effects are largely unknown. This project investigates how GPCRs regulate glucose homeostasis and will evaluate them as potential treatments.
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    Funded Activity

    Oxygen Consumption In Muscle Tissue

    Funder
    National Health and Medical Research Council
    Funding Amount
    $118,768.00
    More information

    Showing 1-10 of 16 Funded Activites

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