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Research Topic : ace genotyping
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  • Funded Activity

    Genetic Influences On Early Onset Coronary Disease

    Funder
    National Health and Medical Research Council
    Funding Amount
    $90,571.00
    More information
    Funded Activity

    ACE Inhibition - A Potntial New Therapy For Peripheral Arterial Disease

    Funder
    National Health and Medical Research Council
    Funding Amount
    $319,488.00
    More information
    Funded Activity

    Tissue Angiotensin And Kinin Systems

    Funder
    National Health and Medical Research Council
    Funding Amount
    $1,032,536.00
    More information
    Funded Activity

    Bayesian Statistical Methods For Combining Evidence In Genetic Association Studies Of Complex Disease.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $471,058.00
    More information
    Funded Activity

    Factors Involved In Maintenance Of Low Malaria Endemicity, And Effects Of Intervention

    Funder
    National Health and Medical Research Council
    Funding Amount
    $88,234.00
    More information
    Funded Activity

    Genetic Variation In IL23R, And Its Role In Ankylosing Spondlysis

    Funder
    National Health and Medical Research Council
    Funding Amount
    $44,440.00
    More information
    Funded Activity

    Influence Of The Genetic Variation In Drug Metabolism On Therapeutic Outcome

    Funder
    National Health and Medical Research Council
    Funding Amount
    $299,085.00
    More information
    Funded Activity

    Studies Of Angiotensin Converting Enzyme In Health And Disease

    Funder
    National Health and Medical Research Council
    Funding Amount
    $101,799.00
    More information
    Funded Activity

    Molecular Evolution And Rapid Typing Of Enteroviruses By Molecular Methods

    Funder
    National Health and Medical Research Council
    Funding Amount
    $44,934.00
    Summary
    Human enteroviruses (HEVs) are common human pathogens associated with a wide spectrum of symptoms ranging from asymptomatic infection to serious illness, especially in infants and the immunocompromised. They are responsible for large outbreaks of diseases. We will develop rapid molecular typing methods for enteroviruses, including reverse line blot hybridization and rolling circle amplification. Meanwhile, we will analyze molecular evolution of important enteroviruses over time.
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    Funded Activity

    Understanding Vasoactive Hormone Pathways In Diabetic Complications

    Funder
    National Health and Medical Research Council
    Funding Amount
    $453,750.00
    Summary
    High blood pressure damages tissues. In clinical practice blood pressure is measured in large arteries, such as the brachial artery in the arm. However, it is the pressure within the organ such as the kidney that actually causes the damage. In particular, the sieving apparatus of the kidney (called the glomerulus), is especially sensitive to the effects of pressure. In diabetes, the pressure within the glomerulus is high because its outflow valve (called the efferent arteriole) is tightly constr .... High blood pressure damages tissues. In clinical practice blood pressure is measured in large arteries, such as the brachial artery in the arm. However, it is the pressure within the organ such as the kidney that actually causes the damage. In particular, the sieving apparatus of the kidney (called the glomerulus), is especially sensitive to the effects of pressure. In diabetes, the pressure within the glomerulus is high because its outflow valve (called the efferent arteriole) is tightly constricted. Therefore even if blood pressure is thought to be normal when measured in the arm, it may still be excessively high within the kidney. Studies have already shown that lowering within-kidney pressure may have a major impact on the progression of kidney disease in diabetes. However, to date this reduction of within-kidney pressure has been sub-maximal. The planned studies will involve the use of new compounds which have more powerful effects in reducing the formation or action of hormones which promote constriction of vessels in the kidney leading to elevated pressure within the kidney. Furthermore, some of these very new agents can open up or dilate these kidney vessels thereby achieving excellent reductions in the pressure inside the kidney. The proposed studies aim to examine new strategies for preferentially lowering pressure within the kidney down to these ideal levels. These hormones also have other effects which could be relevant to non-kidney sites of injury in diabetes including blood vessels and the retina.
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