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Field of Research : Medical Virology
Research Topic : Virus evolution
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  • Funded Activity

    The Phylodynamics Of Human Enteroviruses: Informing Vaccine Effectiveness And Outbreak Preparedness

    Funder
    National Health and Medical Research Council
    Funding Amount
    $564,868.00
    Summary
    Enteroviruses such as EV-A71 are increasing in frequency and in Australia have resulted in the hospitalization of children with severe encephalitis. However, major aspects of their evolution and epidemiology are unknown. We will provide new insights on these important pathogens by revealing which enteroviruses are circulating in the region and whether they arise in specific localities, whether some are commonly associated with severe encephalitis, and whether some pose a greater epidemic threat.
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    Funded Activity

    Host-pathogen Interaction: The Battle For Supremacy

    Funder
    National Health and Medical Research Council
    Funding Amount
    $480,014.00
    Summary
    This grant will provide salary support for Dr Rowena Bull. Dr Bull's research is focused on understanding the disease process between the human host and the infecting virus. The outcomes of this research will be used to find novel ways to fight viral infections with vaccines and drugs.
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    Funded Activity

    Macrophages Drives The Diversity Of HIV

    Funder
    National Health and Medical Research Council
    Funding Amount
    $654,381.00
    Summary
    The diversity of HIV quasispecies within a single AIDS patient is far greater than the global diversity of influeneza annually, highlighting the enormous burden HIV imposes on the immune network. The capacity of HIV-1 to evolve quickly has significantly impaired our effort to produce effective vaccine and long lasting treatment strategy. This project utilizes multidisciplinary approaches to delineate determinants that drives the diversification of HIV-1.
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    Funded Activity

    Resolving Human Immunodeficiency Virus (HIV) Transmission

    Funder
    National Health and Medical Research Council
    Funding Amount
    $745,213.00
    Summary
    To increase the breadth of HIV prevention strategies, it is imperative that we biologically understand how HIV enters our bodies. Through two unique clinical cohorts, we will determine why circumcision is protective and how a commonly acquired sexual transmitted infection (human papilloma virus) can increase HIV transmission.
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    Funded Activity

    Intrinsic Host Antiviral Activity Against Pathogenic Filoviruses

    Funder
    National Health and Medical Research Council
    Funding Amount
    $488,754.00
    Summary
    Bats are a major reservoir for deadly human viruses including Ebola and Marburg virus. In contrast to humans, bats can be infected with these viruses without showing clinical signs of disease. The reason why bats can co-exist with these viruses is unknown. This study will determine if a bat antiviral molecule contributes to limiting virus release compared to the human version that could reveal strategies to prevent and control these deadly viruses in humans.
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    Funded Activity

    Identification Of Host Factors That Restrict Influenza Virus Replication In Macrophages

    Funder
    National Health and Medical Research Council
    Funding Amount
    $566,446.00
    Summary
    Influenza virus infects different cells in the airways, including immune cells (macrophages) and non-immune cells (epithelial cells). Epithelial cell infection results in virus amplification and release whereas macrophage infection leads to virus destruction. This project will identify cellular factors expressed by macrophages that block virus amplification and release. Identification of novel antiviral factors is an important step towards developing strategies to reduce influenza disease.
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    Funded Activity

    Identification Of Host Restriction Factors That Block Respiratory Virus Infection

    Funder
    National Health and Medical Research Council
    Funding Amount
    $956,898.00
    Summary
    Following inhalation, respiratory viruses can infect and grow in airway epithelial cells. Although immune cells such as macrophages are also susceptible to infection, this is generally abortive and new viruses are not released. This project will identify proteins induced in macrophages that block respiratory viruses and prevent their spread in the airways. We will also define mechanisms by which some virulent strains overcome this block to grow in macrophages.
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    Funded Activity

    The Role Of Varicella Zoster Virus In Modulating Cutaneous Infection

    Funder
    National Health and Medical Research Council
    Funding Amount
    $555,892.00
    Summary
    Varicella zoster virus (VZV) causes two skin diseases: chickenpox and shingles. VZV can causes significant morbidity in children and adults and life-threatening disease in immunocompromised people. This project aims to improve our understanding of how VZV affects the function of specialised skin cells to provide information for the development of a better vaccine to lessen the impact of VZV disease on the community.
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    Funded Activity

    Comparative Expression Studies To Identify Cellular Factors Promoting Hendra Virus Replication For A Comprehensive Understanding Of Hendra Virus Pathogenesis

    Funder
    National Health and Medical Research Council
    Funding Amount
    $374,619.00
    Summary
    Hendra virus (HeV) is an emerging pathogen indigenous to fruit bats. HeV is associated with limited outbreaks with high mortality in domesticated animals and humans. To advance the understanding of HeV-related pathogenesis, we will perform comparative studies in bat and human cell lines to recognise differences in virus-host cell interactions leading to a comprehensive understanding of the HeV life cycle and pathogenesis.
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    Funded Activity

    Structure And Function Of The HCV Glycoproteins

    Funder
    National Health and Medical Research Council
    Funding Amount
    $598,863.00
    Summary
    Hepatitis C Virus infects 3% of the world's population causing recurring liver disease, cirrhosis and hepatocellular carcinoma. To infect a liver cell, the viral glycoproteins attach to cell surface molecules wher they are activated to mediate merger of the viral and cellular membranes. This project grant will explore how the viral glycopropteins become activated and obtain essential structural information on the viral glycoproteins. These studies will help us to design antiviral agents.
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