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  • Funded Activity

    EPIGENETIC REPROGRAMMING OF MALIGNANT BREAST CANCER

    Funder
    National Health and Medical Research Council
    Funding Amount
    $863,268.00
    Summary
    Poorly differentiated breast cancers are aggressive tumors, frequently resistant to chemotherapy and associated with high morbidity. Herein we propose the engineering of more selective therapeutic agents able to target the genes involved in cancer initiation and resistance to treatment. We aim to correct and reprogram the cancer cell genome in state that is similar to normal, not tumorigenic cells. This work will generate novel forms of treatment for cancers that are presently not curable.
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    Funded Activity

    Hormonal Regulation Of Growth: Clinical And Molecular Mechanisms

    Funder
    National Health and Medical Research Council
    Funding Amount
    $111,270.00
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    Funded Activity

    Structural And Functional Analysis Of A Cancer-linked Co-regulator Complex

    Funder
    National Health and Medical Research Council
    Funding Amount
    $729,571.00
    Summary
    We seek to understand the mechanisms by which genes are switched on and off throughout our lifetime. A number of multi-component protein machines are involved in this process but their make-up and mechanism of action is not understood. We will investigate the structure and function of one of these machines that has been strongly linked to cancer.
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    Funded Activity

    Post-GWAS Functional Characterisation Of Breast Cancer Susceptibility Loci

    Funder
    National Health and Medical Research Council
    Funding Amount
    $764,632.00
    Summary
    Recent studies have identified regions within the human genome in which DNA sequence variations are associated with an increased risk of breast cancer. Several of these regions do not contain any known genes, suggesting that regulatory DNA sequences are responsible for the associated risk. The aim of this proposal is to identify and characterise these DNA sequences. Understanding how sequences variations in these regions contribute to breast cancer will provide novel avenues for therapy.
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    Funded Activity

    High-throughput Identification And Evaluation Of New Breast Cancer Genes From GWAS.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $841,075.00
    Summary
    Recent studies have identified DNA markers within the human genome that are associated with an increased risk of breast cancer. Most of these markers are located in noncoding regions, therefore the key genes driving risk are not known. This proposal will identify the target genes at all breast cancer risk regions and assess how specific markers affect disease risk. Understanding how DNA variation contributes to breast cancer will provide new avenues for prevention or treatment.
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    Funded Activity

    SARA: Delineating Its Association With The Onset And Development Of Liver Fibrosis

    Funder
    National Health and Medical Research Council
    Funding Amount
    $865,972.00
    Summary
    Liver disease, a significant burden on society, affects many in the prime of their life. Scarring of the liver is a response to injury due to many factors including alcohol, viruses, obesity, and fatty-liver disease. We have identified a protein associated with liver injury. In this project we will perform a systematic analysis to understand the role of this protein in injury progression. Ultimately we intend to develop tools to prevent and treat liver injury.
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    Funded Activity

    Molecular Regulation Of Metabolism And Body Composition By Ski Via Crosstalk With Nuclear Hormone Receptor Signalling.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $558,441.00
    Summary
    Obesity is a common and burdensome health problem in the community which leads to diabetes and heart disease. A number of factors, including hormones play important roles in determing risk of obesity. This study proposes to investigate whether the Ski gene which is a regulatory factor for many hormones affects metabolism in transgenic mouse models of altered Ski function. The proposed studies may identify Ski as a target for therapy for obesity and improvement in sketal muscle metabolism.
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    Funded Activity

    Novel Insights Into The Pathobiology Of Alphavirus Infections

    Funder
    National Health and Medical Research Council
    Funding Amount
    $583,477.00
    Summary
    Ross River virus and chikungunya virus cause muscle and joint pain that can persist for a long time. This project looks at factors in the human host that affect the disease severity, with the aim of finding new treatments.
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    Funded Activity

    Role Of IGF Binding Protein-3 (IGFBP-3) And IGFBP-5 As Modulators Of Nuclear Hormone Signalling

    Funder
    National Health and Medical Research Council
    Funding Amount
    $465,750.00
    Summary
    The insulin-like growth factors are small proteins involved in the growth of most tissues. Their actions are regulated by binding to larger proteins (known as IGFBPs) in the bloodstream and outside the cell. However, some IGFBPs are also found inside cells, where they seem to carry out other functions. We believe that two of these binding proteins, IGFBP-3 and IGFBP-5, change the way cells respond to vitamin A and vitamin D. These two vitamins are important in cell growth and in the way certain .... The insulin-like growth factors are small proteins involved in the growth of most tissues. Their actions are regulated by binding to larger proteins (known as IGFBPs) in the bloodstream and outside the cell. However, some IGFBPs are also found inside cells, where they seem to carry out other functions. We believe that two of these binding proteins, IGFBP-3 and IGFBP-5, change the way cells respond to vitamin A and vitamin D. These two vitamins are important in cell growth and in the way certain cells perform specialised functions. In test-tube experiments, IGFBP-3 and IGFBP-5 interact directly with the receptors that regulate the effects of these hormones. If the same thing happens inside the cell, IGFBP-3 and IGFBP-5 could change the way these receptors respond to signals from outside the cell. We will investigate what effect these IGFBPs have in living cells and in whole animals and how this may relate to human disease. If we are able to understand how IGFBP-3 and IGFBP-5 affect the way cells respond to vitamin A and D, then we may be able to develop new ways to treat certain human diseases.
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