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  • Funded Activity

    Immaturity Of The Human Infant Immune Response: Cellular And Molecular Mechanisms And Clinical Consequences

    Funder
    National Health and Medical Research Council
    Funding Amount
    $308,267.00
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    Funded Activity

    Detection Of Somatic Mutations In Sporadic Epilepsies

    Funder
    National Health and Medical Research Council
    Funding Amount
    $1,256,166.00
    Summary
    Finding genetic causes of epilepsies is essential for refining treatments and genetic counseling. Genetic mutations may occur after fertilization (somatic mutations). These can be difficult to detect by routine genetic tests. We aim to identify somatic mutations by: very deep sequencing of blood to find low concentrations of mutations, analysing DNA from the cerebrospinal fluid, and analysing DNA obtained from the back of the nose which is closely related to brain tissue.
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    Funded Activity

    Somatic Retrotransposition Drives Neoplastic Mutagenesis In Glioblastoma Multiforme

    Funder
    National Health and Medical Research Council
    Funding Amount
    $667,342.00
    Summary
    Retrotransposons are mobile genes that copy-and-paste themselves in our genome. Previously thought to represent “junk DNA”, retrotransposons are increasingly found to play major roles in biology. In a recent landmark publication in Nature, we demonstrated that retrotransposons move in the healthy human brain. In the current study, we will use cutting-edge technologies to determine whether brain cancer can occur as a result. This will provide new perspectives of the genetic basis for cancer.
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    Funded Activity

    The Role Of Somatic Mutations In CCCTC-binding Factor (CTCF) Binding Sites In Cancer

    Funder
    National Health and Medical Research Council
    Funding Amount
    $371,114.00
    Summary
    The three dimensional organisation of genomic DNA has been recognised to play a crucial role in maintaining the stability and function of human cells. In cancer this organisation is often perturbed as a result of mutations to proteins that govern this process. This project will examine how mutations in the DNA may potentially alter the three dimensional organisation of cancer genomes and will identify links between these mutations with cancer development and patient prognosis.
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    Funded Activity

    Identification And Functional Characterisation Of Novel Ovarian Cancer Oncogenes Tumour Suppressor Genes

    Funder
    National Health and Medical Research Council
    Funding Amount
    $452,234.00
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    Funded Activity

    Consequences Of MYD88 Mutations Commonly Found In Human B Cell Malignancies

    Funder
    National Health and Medical Research Council
    Funding Amount
    $442,583.00
    Summary
    MYD88 is one of the most recurrently mutated genes in B cell malignancies, such as diffuse-large B cell lymphoma and Waldenström macroglobulinemia. This project will characterise oncogenic MYD88 mutations by introducing the mutations into normal mouse B cells. It will examine how the mutations disrupt signalling pathways and B cell functions and how the mutations respond to new lymphoma drugs. We hope this project will provide information for lymphoma pathogenesis and rational drug selection.
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    Funded Activity

    Understanding Causes Of The Rising Incidence Of Thyroid Cancer – What Can Mutations In The BRAF Oncogene Tell Us About Causes And Diagnostic Pathways For Thyroid Cancer?

    Funder
    National Health and Medical Research Council
    Funding Amount
    $610,222.00
    Summary
    The occurrence of thyroid cancer has increased rapidly over the last 25 years but the cause is unknown. The increase may reflect ‘over-diagnosis’ of less harmful cancers or greater exposure to causes of this cancer. Evidence suggests that a gene mutation (BRAF) in thyroid cancers is important in understanding the drivers of the increase. This study will examine the increase in thyroid cancer by investigating causes and diagnostic pathways considering the presence or absence of BRAF mutations.
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    Funded Activity

    Immunoglobulin Germline Genes, BCR Repertoire Development And Disease Susceptibility. An Investigation Of Haplotypic Variation Between Individuals

    Funder
    National Health and Medical Research Council
    Funding Amount
    $519,828.00
    Summary
    The immune system is capable of making a repertoire of protective antibodies including literally tens of millions of different specificities. These are produced by permutations and combinations of a small set of ‘germline’ genes. This project will analyse how individual variations in the germline genes lead to individual differences in the repertoires of available antibodies, and will investigate whether or not such differences contribute to our susceptibility to infection and autoimmune disease .... The immune system is capable of making a repertoire of protective antibodies including literally tens of millions of different specificities. These are produced by permutations and combinations of a small set of ‘germline’ genes. This project will analyse how individual variations in the germline genes lead to individual differences in the repertoires of available antibodies, and will investigate whether or not such differences contribute to our susceptibility to infection and autoimmune diseases.
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    Funded Activity

    PRECISION: Personalised Risk Evaluation In DCIS, International

    Funder
    National Health and Medical Research Council
    Funding Amount
    $1,392,930.00
    Summary
    Ductal carcinoma in situ (DCIS) of the breast is a common diagnosis with problematic clinical management. This study brings together an international consortium to identify and validate clinical biomarkers of recurrence.
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    Funded Activity

    Physiologic And Aberrant DNA Recombination In B Lymphocytes

    Funder
    National Health and Medical Research Council
    Funding Amount
    $408,768.00
    Summary
    B cells produce antibody which is critical to fight infection. In order to perform this function, antibody genes must first be modified by immune enzymes. However, abnormal DNA attack by these enzymes outside of antibody genes can result in B cell cancer. How the immune system detects and destroys cancerous B cells is poorly understood. This research will provide insight into these processes, and in doing so will further our understanding of how B cell cancers develop and how they are destroyed.
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