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Research Topic : SIGNALLING PATHWAYS
Status : Closed
Field of Research : Cellular Immunology
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Cellular Immunology (23)
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  • Funded Activity

    Dendritic Cell-mediated Induction Of T Cell Tolerance

    Funder
    National Health and Medical Research Council
    Funding Amount
    $654,725.00
    Summary
    Australia has some of the highest rates of immune-mediated diseases in the world. These diseases include autoimmune, allergic and inflammatory conditions. We will use a mouse model to study how dendritic cells can prevent the onset of these conditions by inactivating the immune cells that cause them. Our findings will aid in understanding why these diseases develop and how they may be prevented and treated.
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    Funded Activity

    Investigation Of Cellular Abnormalities And Synapse Formation In DOCK8 Immunodeficiency

    Funder
    National Health and Medical Research Council
    Funding Amount
    $318,284.00
    Summary
    Why do some people get allergies? Or serious infections? To investigate this we will study mice and humans with a mutation in the DOCK8 gene. People with mutations in the DOCK8 gene get Hyper-IgE Syndrome and develop severe viral infections of the skin as well as allergic disease. By investigating how DOCK8 works in the cells of the immune system, we hope to understand why these infections and allergies occur and find out why these problems can also happen in those without this specific genetic .... Why do some people get allergies? Or serious infections? To investigate this we will study mice and humans with a mutation in the DOCK8 gene. People with mutations in the DOCK8 gene get Hyper-IgE Syndrome and develop severe viral infections of the skin as well as allergic disease. By investigating how DOCK8 works in the cells of the immune system, we hope to understand why these infections and allergies occur and find out why these problems can also happen in those without this specific genetic defect.
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    Funded Activity

    Taking The First Steps From Promise To Product: Exploration Of The Newly Discovered Interleukin 37 Receptor Complex And Its Signaling Pathways

    Funder
    National Health and Medical Research Council
    Funding Amount
    $694,623.00
    Summary
    Cytokines are messenger proteins used by most cells of the body. They function as master regulators of many biological processes and as such play an important role in a wide spectrum of diseases. Anti-inflammatory cytokines attenuate the potentially destructive force of other cytokines and the immune system as a whole, and are therefore coveted as drug targets. We discovered the formidable anti-inflammatory properties of IL-37 and will now explore how these activites can be utilised in clinical .... Cytokines are messenger proteins used by most cells of the body. They function as master regulators of many biological processes and as such play an important role in a wide spectrum of diseases. Anti-inflammatory cytokines attenuate the potentially destructive force of other cytokines and the immune system as a whole, and are therefore coveted as drug targets. We discovered the formidable anti-inflammatory properties of IL-37 and will now explore how these activites can be utilised in clinical medicine.
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    Funded Activity

    Immune Imprinting By Nanoparticles And Vaccines: New Principles And Translation Into The Clinic

    Funder
    National Health and Medical Research Council
    Funding Amount
    $631,010.00
    Summary
    Vaccines require adjuvants to be effective. Despite decades of research there is only one adjuvant approved for broad use in humans. Based on our prior findings I will engage new principles in nanotechnology, and deepen understanding of immune imprinting in various organs of the body including the lung, to develop 2nd generation broadly useful nanoadjuvants able to effectively treat cancer and malaria.
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    Funded Activity

    Roles Of Signalling In The Control Of Immune System Development, Function And Pathology

    Funder
    National Health and Medical Research Council
    Funding Amount
    $737,936.00
    Summary
    This research focuses on the function of the NF-?B and MAP kinase biochemical pathways in immune cells. Both pathways regulate gene expression controlling the development, division, viability and function of immune cells. Consistent with these roles, impaired regulation of these pathways contributes to many immune related diseases. My goal is to utilize information learnt about these pathways and apply it to developing therapies for treating diseases afflicting the immune system.
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    Funded Activity

    Modulation Of T Cell Surface Receptors That Are Dependent On Extrinsic Tyrosine Kinases.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $372,009.00
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    Funded Activity

    Investigating B Cell Development, Maintenance And High-affinity Antibody Production By ENU Mutagenesis

    Funder
    National Health and Medical Research Council
    Funding Amount
    $408,388.00
    Summary
    B cells are essential for the protection against infections. This application aims to identify new genes that are crucial for the development or function of B cells and will investigate how mutations in newly discovered genes contribute to defects in the development and function of B cells and the pathogenesis of B cell leukaemia.
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    Funded Activity

    Determining The Role Of Rel/NF-kB Transcription Factors In CD8 T Cell Homeostasis.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $426,500.00
    Summary
    NF-kB proteins comprise a family of transcription factors that regulate key genes involved in immune responses, inflammation, cell death and proliferation. This family of proteins are potential drug targets for treatment of various diseases. How and when such inhibitors are used in clinical situations depends on understanding how and which cells of the immune system are specifically affected by the absence of NF-kB proteins. In a number of treatment settings intercurrent viral infections occur f .... NF-kB proteins comprise a family of transcription factors that regulate key genes involved in immune responses, inflammation, cell death and proliferation. This family of proteins are potential drug targets for treatment of various diseases. How and when such inhibitors are used in clinical situations depends on understanding how and which cells of the immune system are specifically affected by the absence of NF-kB proteins. In a number of treatment settings intercurrent viral infections occur frequently and therefore there is an even greater need to understand how the immune system may be affected or compromised in response to the primary treatment. This work will provide insights into the cellular and molecular mechanisms affected by the absnece of a particular NF-kB family member (NF-kB1) in CD8 T cells during normal T cell homeostasis and when challenged with viruses. What we learn from our experiments could have important implications for the development of vaccines.
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    Funded Activity

    Elucidating Immune Responses By Single Cell Pedigree And Tracing Analysis

    Funder
    National Health and Medical Research Council
    Funding Amount
    $666,950.00
    Summary
    To develop vaccines and to combat autoimmunity, we need to understand how initial immune activation influences the fate of immune cells and their progeny. To achieve this, we have developed microscopic techniques and analytical software with which to observe how initial signalling processes in the parent immune cell influence the death, proliferation and differentiation of its daughters, granddaughters and further progeny. We will use these approaches to determine how immune cell fate is control
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    Funded Activity

    How Does T Cell Receptor Signaling Begin?

    Funder
    National Health and Medical Research Council
    Funding Amount
    $318,768.00
    Summary
    The initial step of T cell activation of how the external ligand binding is translated to an increase of receptor phosphorylation at the cytoplasmic side is remain poorly understood. It is believed that the loss of immune recognition in cancer and over reactivity in auto-immune diseases are caused by abnormality of this transmembrane signalling transduction. Clarification of this molecular machinery can provide a molecular basis of those diseases and guidelines of more effective therapies.
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    Showing 1-10 of 23 Funded Activites

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