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Research Topic : Retinal function
Country : Australia
Scheme : Project Grants
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  • Funded Activity

    A Fibroin-based Prosthetic Bruch's Membrane For The Treatment Of Age-related Macular Degeneration

    Funder
    National Health and Medical Research Council
    Funding Amount
    $538,080.00
    Summary
    Our aim is to develop a new therapy for the treatment of patients with age-related macular degeneration (AMD), a leading cause of blindness in our ageing population. The novelty of our therapy resides in using a protein derived from silk fibers (fibroin), to rebuild a healthy barrier between the outermost layer of the retina and adjacent blood vessels. We expect that the findings from this study will eventually lead to better outcomes for patients with AMD.
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    Funded Activity

    The Role Of Microglia In Early Diabetic Retinopathy

    Funder
    National Health and Medical Research Council
    Funding Amount
    $665,582.00
    Summary
    Diabetic retinopathy is one of the most feared complications of diabetes. This project will examine the role that retinal immune cells called microglia have in causing early changes in the vasculature. We will examine whether diabetes changes the way neurons communicate with blood vessels, opening up a possible treatment target that could prevent the progression to more advanced disease.
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    Funded Activity

    Self-destructing CRISPR-constructs For Targeted Genome Editing In The Retina.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $679,926.00
    Summary
    Despite the identification of specific mutations causing many inherited retinal dystrophies, all of these conditions are currently untreatable. We have established gene-editing techniques and have developed a novel mouse model, which will serve as a robust platform for testing different techniques of gene editing in the retina. No other group in the world is known to be using this platform for gene editing and our work will expedite the clinical translation of this technology.
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    Funded Activity

    The Structural Basis For Glutamate Transporter Function

    Funder
    National Health and Medical Research Council
    Funding Amount
    $373,144.00
    Summary
    Glutamate transporters are vacuum cleaners in the brain that suck the neurotransmitter glutamate into cells. When the glutamate vacuum breaks down or becomes blocked, glutamate levels outside cells increase, leading to cell death in the brain. This process underlies the damage in many brain diseases including Alzheimer’s disease and stroke. The aim of this project is to understand the mechanism of the glutamate vacuum cleaner so we can develop therapeutics to fix it when it breaks down.
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    Funded Activity

    Signaling Pathways To Enhance Potency Of AMPK-targeting Drugs

    Funder
    National Health and Medical Research Council
    Funding Amount
    $661,966.00
    Summary
    Sedentary lifestyles and consumption of high energy foods has led to epidemics of obesity-related metabolic diseases that place enormous financial and medical burden on the Australian economy. An attractive drug target to treat these diseases is AMP-activated protein kinase (AMPK) which functions as both a cellular fuel gauge and co-ordinator of whole-body metabolism. Our goal is to improve AMPK drug potency by identifying novel processes that sensitize AMPK to drugs.
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    Funded Activity

    Do Exposures Before Conception Influence The Risk Of Asthma In Offspring?

    Funder
    National Health and Medical Research Council
    Funding Amount
    $688,586.00
    Summary
    Asthma and poor lung function are major causes of public health issues. Emerging evidence suggests adverse exposures even before the conception of a child may cause these conditions. The proposed project is part of an international study across generations to identify these factors. This study will provide novel evidence to guide interventions and identify studies to advance this area further. These original findings will be of great importance both nationally and internationally.
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    Funded Activity

    Clinical Trial Of A Suprachoroidal Visual Prosthesis For The Profoundly Vision Impaired

    Funder
    National Health and Medical Research Council
    Funding Amount
    $1,098,802.00
    Summary
    For 15 years we have been designing a bionic eye. We have made a device called the Phoenix99 and shown in short term animal tests that it is both safe to implant but also that it potentially performs better than any other device in the world. We are requesting funds to complete longer term animal testing of the device and then commence a small human clinical trial to demonstrate the benefits of the technology – specifically that it is able to help blind people navigate without assistance.
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    Funded Activity

    Regulation Of Receptors That Control Platelet Function Under Shear Stress

    Funder
    National Health and Medical Research Council
    Funding Amount
    $507,273.00
    Summary
    Specialized human blood cells that control blood loss and clotting (platelets) are currently difficult to test in the clinical laboratory, meaning patients are at risk of excessive bleeding or serious clot formation during disease or treatment. The aim of this proposal is to use our new reagents and assays to develop more reliable methods for evaluating relative bleeding or clotting risk in individuals.
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    Funded Activity

    Pathobiology That Causes Fatal Thrombosis In HIT

    Funder
    National Health and Medical Research Council
    Funding Amount
    $398,371.00
    Summary
    Autoimmune-based thrombocytopenia can be a life-threatening adverse event associated with viral load, surgery, drug therapies or the use of the anticoagulant, heparin. This grant will define mechanisms of anti-platelet antibody-dependent platelet activation and assess shedding of platelet-specific glycoprotein (GP)VI as an immediate consequence of this activation, provide a new strategy for evaluating risk of thrombosis in HIT.
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    Funded Activity

    The Role Of Duffy And PF4 In The Platelet Killing Of Malaria Parasites.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $350,045.00
    Summary
    Platelets in the blood can kill the Plasmodium parasite, which lives inside red blood cells and causes malaria. Platelets bind parasite-infected red cells and release a molecule that is toxic to the parasite. This project will study why a red cell molecule called Duffy is also needed for this function of platelets. Most Africans carry a gene for Duffy that stops its expression in red cells, and may therefore be more susceptible to malaria because their platelets cannot kill the malaria parasite.
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    Showing 1-10 of 41 Funded Activites

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