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Research Topic : RNA-binding protein
Field of Research : Medical Virology
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  • Funded Activity

    The Role Of Noncoding Subgenomic Flavivirus RNA In Virus-host Interactions

    Funder
    National Health and Medical Research Council
    Funding Amount
    $624,429.00
    Summary
    Flaviviruses such as Dengue, Japanese encephalitis , and West Nile are major human pathogens causing more than 50 million infections per year. Elements in viral genome responsible for pathogenesis of these viruses are not well defined. Recently we have identified a unique for these viruses noncoding subgenomic flavivirus RNA (sfRNA) and showed that it is contributing to viral pathogenesis. In this proposal we aim to determine mechanisms by which sfRNA facilitates viral pathogenesis.
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    Funded Activity

    What Is The Role Of Codon Bias In HIV-1 Genome?

    Funder
    National Health and Medical Research Council
    Funding Amount
    $341,250.00
    Summary
    Both human and viral genetic materials (ribonucleic acids, RNA) are made up of 4 different basic residues, namely A, U, G and C. Combination of any three of these ribonucleic acids residues is known as codon , which is essential to target one of the twenty amino acids to the host cell machinery for the making of proteins. Eighteen out of these twenty amino acids can be represented by more than one codon during the making of proteins. Interestingly, human and viral proteins, such as HIV-1, utilis .... Both human and viral genetic materials (ribonucleic acids, RNA) are made up of 4 different basic residues, namely A, U, G and C. Combination of any three of these ribonucleic acids residues is known as codon , which is essential to target one of the twenty amino acids to the host cell machinery for the making of proteins. Eighteen out of these twenty amino acids can be represented by more than one codon during the making of proteins. Interestingly, human and viral proteins, such as HIV-1, utilise two completely different subsets of codons (codon bias) for the synthesis of their respective proteins. The objective of this proposal is to delineate the functional requirement of this codon bias in HIV-1 replication cycle. Results from this work will identify novel elements that may be used for the design of novel antiretroviral strategy. Furthermore, lesson learned from this project will also provide important clues to improve the efficacy and safety of the design of current retroviral gene delivery vector.
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    Funded Activity

    The Role Of TAR RNA In HIV-1 Reverse Transcription

    Funder
    National Health and Medical Research Council
    Funding Amount
    $76,723.00
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    Funded Activity

    Pathogensis Of Macrophage Tropic HIV-1

    Funder
    National Health and Medical Research Council
    Funding Amount
    $359,250.00
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    Funded Activity

    Cell Surface Lectin Receptors For Attachment And Entry Of Influenza Viruses Into Cells Of The Innate Immune System

    Funder
    National Health and Medical Research Council
    Funding Amount
    $530,094.00
    Summary
    Influenza virus is a leading cause of respiratory infection and death worldwide. Infection of humans is initiated when the virus contacts cells lining the respiratory tract. Infection of epithelial cells leads to virus amplification whereas infection of immune cells results in virus destruction. Despite extensive research efforts, it is not clear how the virus infects these cells. This project aims to identify receptors on human cells used by influenza virus to attach to and infect immune cells.
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    Funded Activity

    Structual Analysis Of Interactions Between HCV E2 Glycoprotein, Scavenger Receptor Class B Type I (SR-BI), And CD81

    Funder
    National Health and Medical Research Council
    Funding Amount
    $299,445.00
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    Funded Activity

    Novel Antivirals For The Treatment Of Hendravirus Infection.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $199,227.00
    Summary
    Hendravirus outbreaks have become frequent and 7 human cases have been reported, this has resulted in 4 deaths. Currently we have no treatment options. Researchers at Griffith University and the CSIRO have developed a new treatment that attacks the virus by turning off the viral genes at the site of infection. The plan is to treat patients soon after infection to slow or stop the virus and allow patients to recover naturally from this highly lethal disease.
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    Funded Activity

    Rhinovirus Protease Subcellular Trafficking And Host Cell Targets; Relevance To Asthma Exacerbation And Vaccine Approaches

    Funder
    National Health and Medical Research Council
    Funding Amount
    $582,072.00
    Summary
    Rhinovirus (RV) infections are the major cause of virus induced asthma attacks, causing significant morbidity and mortality. Asthma & asthma exacerbations are increasing worldwide with new strategies urgently needed to reduce RV-associated disease. We aim to build on our substantive new data, using cutting edge technology to identify new targets for novel asthma therapies.
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    Funded Activity

    Stealth Liposomes And SiRNA For The Treatment Of Respiratory Viral Infections

    Funder
    National Health and Medical Research Council
    Funding Amount
    $528,793.00
    Summary
    Respiratory infections caused by Influenza and Respiratory syncytial virus cause significant hospitalisations and deaths within the community. For example, RSV causes around 1000 hospital admissions of young children a year and there is no cure or vaccination. Therapies are limited and toxic. We will develop and test a novel therapy based on gene silencing to specifically target viral genes, and combine this with our novel drug delivery system for better treatment of these diseases.
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    Funded Activity

    The Role Of Noncoding Viral RNAs In Flavivirus Infection And Exosomal Signalling

    Funder
    National Health and Medical Research Council
    Funding Amount
    $683,447.00
    Summary
    The application is aimed at investigating the novel role for viral noncoding RNAs in exosomal antiviral signalling and associated outcome of infection with West Nile virus. We will identify host enzymes involved in generation of viral noncoding RNAs, determine which host proteins they interact with and how these interactions determine their incorporation into secreted exosomes to influence outcome of infection.
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    Showing 1-10 of 35 Funded Activites

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