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Research Topic : Protein interactions
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  • Funded Activity

    Unraveling The Dynamic Munc18a:Syntaxin1 Interaction Required For Neurotransmission

    Funder
    National Health and Medical Research Council
    Funding Amount
    $674,591.00
    Summary
    Membrane trafficking, the topic of the 2013 Nobel prize in Medicine, is required for delivery of cellular cargo. This research will investigate the interactions and structures of proteins from the neuronal membrane trafficking system. Understanding how this system operates will expand our knowledge of processes fundamental to learning and memory and may ultimately lead to development of selective therapeutics for treating a range of diseases.
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    Funded Activity

    Design And Engineering Of Adnectins For Diagnosis And Therapy

    Funder
    National Health and Medical Research Council
    Funding Amount
    $803,152.00
    Summary
    This project aims to engineer a naturally-occurring human protein, called an adnectin, to produce molecules that are able to bind specific targets in the human body, and as such may be used in the diagnosis and therapy of a range of diseases.
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    Funded Activity

    Characterization Of The Type IX Secretion System In Porphyromonas Gingivalis

    Funder
    National Health and Medical Research Council
    Funding Amount
    $831,656.00
    Summary
    Periodontitis is associated with the keystone pathogen Porphyromonas gingivalis. We have identified a novel protein secretion machine comprised of at least 12 components in P. gingivalis which transports the bacterium's major virulence factors to the cell surface and attaches them to the outer membrane. We aim to determine the spatial arrangement and specific role of each of these 12 components and thereby provide targets for future treatments against this disease.
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    Funded Activity

    Function And Molecular Mechanism Of Histidine-rich Glycoprotein In Necrotic Cell And Pathogen Clearance

    Funder
    National Health and Medical Research Council
    Funding Amount
    $525,957.00
    Summary
    This research proposal is to investigate the molecular mechanism and function of a blood serum protein, histidine-rich glycoprotein (HRG), in protecting against tissue injury caused by inflammation and infection. HRG has been implicated in controlling important aspects of tissue injury by aiding removal of dead cells and pathogens. Understanding the role of HRG in these disease settings may allow the development of approaches for the treatment of inflammatory, autoimmune and infectious disease.
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    Funded Activity

    The Pharmacology Of Sulfotransferase 4A1

    Funder
    National Health and Medical Research Council
    Funding Amount
    $576,966.00
    Summary
    SULT4A1 is not a sulfotransferase, but a sulfotransferase inhibitor. It forms high affinity heterodimers with other sulfotransferases via a conserved dimerisation site in its carboxyl terminus attenuating catalytic activity. Consequently, it is important for the metabolism of numerous important molecules including estrogens, thyroid hormones, neurotransmitters and many therapeutic agents.
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    Funded Activity

    Coupling The Cell Cortex To Membranes: Structural Basis For The Activation And Control Of Ezrin

    Funder
    National Health and Medical Research Council
    Funding Amount
    $587,548.00
    Summary
    Cells are dynamic: they change shape, communicate with each other and import/export signalling molecules. These dynamic processes are controlled via the interaction of the cell membrane with the underlying actin cytoskeleton and they are important for health, for example, they are critical for proper immune cell function. The goal of this project in to unravel the control of membrane dynamics by defining the interactions between the cell membrane and the proteins: ezrin and RhoA.
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    Funded Activity

    How Do BET Bromodomain Proteins Regulate Gene Expression?

    Funder
    National Health and Medical Research Council
    Funding Amount
    $586,791.00
    Summary
    This project is aimed at defining the biochemical mechanisms of action of a class of gene regulatory proteins (BET proteins) that are currently considered to be exciting drug targets for a range of diseases, predominantly cancer. A better understanding of the means by which BET proteins regulate gene expression will be important for the rational design and application of drugs that selectively target the proteins.
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    Funded Activity

    Location, Location, Location: Sub-cellular Specific Targeting Of JNK As A Novel Therapy In Breast Cancer.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $633,755.00
    Summary
    The ‘triple negative’ breast cancer subtype is the most aggressive form of breast cancer, and unlike other subtypes, there are no drugs to specifically this subtype. While many potential drug targets have been identified, they cannot be utilised clinically because of other beneficial roles within the body. We are now deploying our innovative experimental platforms to specifically target the tumour promoting functions of a protein known as ‘JNK’, whilst retaining its beneficial functions.
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    Funded Activity

    Diabolic Regulation Of Macrophage Cell Death Pathways By Legionella

    Funder
    National Health and Medical Research Council
    Funding Amount
    $616,912.00
    Summary
    The bacterial pathogen Legionella causes fatal pneumonia in immuno-compromised humans. Infections depend on a sophisticated secretion machinery that translocates hundreds of proteins into host cells. These proteins subvert several essential defense pathways, including cell death signals. This project will highlight how Legionella interfere with cell death pathways and control the survival of its host cells. These findings will facilitate the development of promising new anti-bacterial agents.
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    Funded Activity

    Endosomal Sorting Of Amyloid Precursor Protein In Alzheimer's Disease

    Funder
    National Health and Medical Research Council
    Funding Amount
    $858,643.00
    Summary
    Alzheimer's Disease is a progressive neurological disorder and is the most common cause of dementia. Effective treatments are desperately needed, but none are currently available. The toxic amyloid peptide A? is central to disease pathology and is derived from breakdown of the Alzheimer’s amyloid precursor protein (APP). In this project we will examine the interactions between APP and the molecular machinery that controls its location in the cell and subsequent degradation.
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    Showing 1-10 of 313 Funded Activites

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