ARDC Research Link Australia Research Link Australia   BETA Research
Link
Australia
  • ARDC Newsletter Subscribe
  • Contact Us
  • Home
  • About
  • Feedback
  • Explore Collaborations
  • Researcher
  • Funded Activity
  • Organisation
  • Researcher
  • Funded Activity
  • Organisation
  • Researcher
  • Funded Activity
  • Organisation

Need help searching? View our Search Guide.

Advanced Search

Current Selection
Field of Research : Protein Targeting And Signal Transduction
Research Topic : MUTAGENESIS
Clear All
Filter by Field of Research
Protein Targeting And Signal Transduction (6)
Genetic Technologies: Transformation, Site-Directed Mutagenesis, Etc. (4)
Biochemistry and Cell Biology (2)
Autoimmunity (1)
Biochemistry And Cell Biology Not Elsewhere Classified (1)
Cell Metabolism (1)
Immunology (1)
Medical Microbiology (1)
Medical Parasitology (1)
Filter by Socio-Economic Objective
Biological sciences (1)
Health related to ageing (1)
Infectious diseases (1)
Inherited diseases (incl. gene therapy) (1)
Other (1)
Sheep—meat (1)
Sheep—wool (1)
Filter by Funding Provider
Australian Research Council (4)
National Health and Medical Research Council (2)
Filter by Status
Closed (6)
Filter by Scheme
Discovery Projects (3)
NHMRC Project Grants (2)
Linkage Projects (1)
Filter by Country
Australia (5)
Filter by Australian State/Territory
VIC (5)
QLD (1)
  • Researchers (11)
  • Funded Activities (6)
  • Organisations (14)
  • Funded Activity

    Structural Studies On Cell Signalling Via The LIF Receptor And Gp130

    Funder
    National Health and Medical Research Council
    Funding Amount
    $453,943.00
    Summary
    The cytokines play important roles in the immune system during blood cell development and inflammation, and in nerve growth, bone remodeling, reproduction and heart development. Cell responses are initiated by a cytokine bringing together on the cell surface a receptor complex made up of multiple molecules. This project will investigate the atomic structure of the cell surface macromolecular complex, and hence the underlying mechanism by which cytokine signals are initiated.
    More information
    Funded Activity

    Regulation Of PtdIns(3,4,5)P3 By Inositol Polyphosphate 5-phosphatases

    Funder
    National Health and Medical Research Council
    Funding Amount
    $200,880.00
    Summary
    Growing cells respond to growth factors by dividing and proliferating. Uncontrolled cell growth leads to cancer. Signals are released from the cell membrane following growth factor stimulation, that communicate via a complex network of intracellular signalling molecules, that instruct the nucleus to divide. One critical signalling network that mediates cell growth are the phosphoinositide messenger molecules. These signals are switched off by a family of proteins called inositol polyphosphate 5- .... Growing cells respond to growth factors by dividing and proliferating. Uncontrolled cell growth leads to cancer. Signals are released from the cell membrane following growth factor stimulation, that communicate via a complex network of intracellular signalling molecules, that instruct the nucleus to divide. One critical signalling network that mediates cell growth are the phosphoinositide messenger molecules. These signals are switched off by a family of proteins called inositol polyphosphate 5-phosphatases. We propose the 5-phosphatases are essential for normal cell growth. Several studies have suggested in their absence tumour formation may occurr. We have identified a new member of this enzyme family called SHIP-2. This proposal aims to investigate the mechanisms by which this enzyme family metabolises signalling molecules and thereby regulates cell growth. We will also characterize how the 5-phosphatases control the normal pathways by which primitive cells differeniate into mature cells.
    Read more Read less
    More information
    Funded Activity

    Discovery Projects - Grant ID: DP0208165

    Funder
    Australian Research Council
    Funding Amount
    $695,000.00
    Summary
    Post-genomic investigation of the relict plastid and mitochondrion of malaria parasites. Malaria is a major global health problem. The malaria parasite has two substructures, a relict chloroplast and a mitochondrion, that are excellent targets for new and existing drugs. However, we do not know the key functions of these two compartments. The entire genetic blueprint (genome) is now available for the malaria parasite and I propose to determine exactly which parts of the genome service the rel .... Post-genomic investigation of the relict plastid and mitochondrion of malaria parasites. Malaria is a major global health problem. The malaria parasite has two substructures, a relict chloroplast and a mitochondrion, that are excellent targets for new and existing drugs. However, we do not know the key functions of these two compartments. The entire genetic blueprint (genome) is now available for the malaria parasite and I propose to determine exactly which parts of the genome service the relict chloroplast and mitochondria. This will sketch out a picture of their inner workings. Armed with this information we can take a rational approach to seeking an Achilles? Heel of malaria against which parasite-specific drugs can be developed.
    Read more Read less
    More information
    Funded Activity

    Linkage Projects - Grant ID: LP0991919

    Funder
    Australian Research Council
    Funding Amount
    $336,000.00
    Summary
    Inhibition of pro-inflammatory cytokine secretion- A new route to therapeutics of chronic inflammatory disease. Chronic inflammatory diseases, including rheumatoid arthritis and inflammatory bowel disease, affect millions of people leading to considerable suffering, economic loss and premature death. Anti-TNF treatments have recently shown success in the treatment of rheumatoid arthritis, inflammatory bowel disease and other conditions, however, a substantial number of patients (~50%) do not re .... Inhibition of pro-inflammatory cytokine secretion- A new route to therapeutics of chronic inflammatory disease. Chronic inflammatory diseases, including rheumatoid arthritis and inflammatory bowel disease, affect millions of people leading to considerable suffering, economic loss and premature death. Anti-TNF treatments have recently shown success in the treatment of rheumatoid arthritis, inflammatory bowel disease and other conditions, however, a substantial number of patients (~50%) do not respond to the current TNF treatments. Improved anti-TNF strategies would provide enhanced health outcomes and welcome relief to many Australians. In addition, the economic benefit of the TNF market is very substantial. Therefore the potential impact of this research is very high both for health care and economical potential.
    Read more Read less
    More information
    Funded Activity

    Discovery Projects - Grant ID: DP0208857

    Funder
    Australian Research Council
    Funding Amount
    $180,000.00
    Summary
    Shaping a signal: studies on non-contiguous residues in an intracellular serpin that constitute a novel nuclear protein import signal. Eukaryotic cells contain membrane-bound organelles like the nucleus, endoplasmic reticulum and mitochondria, and use specific mechanisms to direct proteins from their site of synthesis to their target organelle. In nuclear proteins, sequence motifs termed nuclear localization signals (NLSs) direct engagement with the nuclear pore complex and translocation from cy .... Shaping a signal: studies on non-contiguous residues in an intracellular serpin that constitute a novel nuclear protein import signal. Eukaryotic cells contain membrane-bound organelles like the nucleus, endoplasmic reticulum and mitochondria, and use specific mechanisms to direct proteins from their site of synthesis to their target organelle. In nuclear proteins, sequence motifs termed nuclear localization signals (NLSs) direct engagement with the nuclear pore complex and translocation from cytoplasm to nucleus. All NLSs described so far consist of 5-7 contiguous basic residues. We propose to study a novel NLS that we recently discovered on an intracellular serpin. This comprises non-contiguous residues that together form a basic "patch" on the mature protein, and is the first example of a conformational NLS.
    Read more Read less
    More information
    Funded Activity

    Discovery Projects - Grant ID: DP0208773

    Funder
    Australian Research Council
    Funding Amount
    $180,000.00
    Summary
    The Molecular Basis of Copper Metabolism in Sheep. The unusual copper metabolism of sheep represents a significant agricultural problem. They are very susceptible to copper deficiency, but readily accumulate copper to toxic levels in the liver leading to fatal liver failure. We propose to elucidate the reason for the copper accumulation phenotype of sheep. We are focussing on WND, a copper transporter responsible for copper excretion into bile. We discovered a novel form of sheep WND designated .... The Molecular Basis of Copper Metabolism in Sheep. The unusual copper metabolism of sheep represents a significant agricultural problem. They are very susceptible to copper deficiency, but readily accumulate copper to toxic levels in the liver leading to fatal liver failure. We propose to elucidate the reason for the copper accumulation phenotype of sheep. We are focussing on WND, a copper transporter responsible for copper excretion into bile. We discovered a novel form of sheep WND designated WNDb to distinguish it from the normal form, WNDa. The experiments outlined are designed to understand the function of both proteins in the sheep and their role in copper sequestration.
    Read more Read less
    More information

    Showing 1-6 of 6 Funded Activites

    Advanced Search

    Advanced search on the Researcher index.

    Advanced search on the Funded Activity index.

    Advanced search on the Organisation index.

    National Collaborative Research Infrastructure Strategy

    The Australian Research Data Commons is enabled by NCRIS.

    ARDC CONNECT NEWSLETTER

    Subscribe to the ARDC Connect Newsletter to keep up-to-date with the latest digital research news, events, resources, career opportunities and more.

    Subscribe

    Quick Links

    • Home
    • About Research Link Australia
    • Product Roadmap
    • Documentation
    • Disclaimer
    • Contact ARDC

    We acknowledge and celebrate the First Australians on whose traditional lands we live and work, and we pay our respects to Elders past, present and emerging.

    Copyright © ARDC. ACN 633 798 857 Terms and Conditions Privacy Policy Accessibility Statement
    Top
    Quick Feedback