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Research Topic : INTRACELLULAR SIGNAL
Scheme : NHMRC Project Grants
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  • Funded Activities (288)
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  • Funded Activity

    Mechanism Of Release Of Surfactant

    Funder
    National Health and Medical Research Council
    Funding Amount
    $259,170.00
    More information
    Funded Activity

    The Integration Of The JAK/STAT Pathway Into The Network Of Signal Transduction Pathways In The Cell.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $192,473.00
    More information
    Funded Activity

    The Role Of Signal Terminating Enzymes In Cell Activati On

    Funder
    National Health and Medical Research Council
    Funding Amount
    $295,162.00
    More information
    Funded Activity

    The Regulation Of PI 3-kinase And 3-position Phosphoinositides

    Funder
    National Health and Medical Research Council
    Funding Amount
    $312,656.00
    More information
    Funded Activity

    The Regulation Of A Novel Molecule Involed In Cellular Activation

    Funder
    National Health and Medical Research Council
    Funding Amount
    $145,532.00
    More information
    Funded Activity

    Investigation Of A Novel Enzyme Regulating Platelet Fun Ction

    Funder
    National Health and Medical Research Council
    Funding Amount
    $77,735.00
    More information
    Funded Activity

    Roles Of Chaperone Proteins And Phosphorylation Events In Activity Of The Dioxin Receptor

    Funder
    National Health and Medical Research Council
    Funding Amount
    $227,036.00
    Summary
    Polycyclic aromatic hydrocarbons (PAHs) are prevalent environmental pollutants which cause a wide range of deleterious health effects. Metabolic activation of PAHs occurs primarily through a set of intracellular oxidising enzymes which are induced by the presence of PAHs. The dioxin receptor is a gene regulatory protein that is pivotal in the metabolic pathway as it links the presence of contaminating PAHs to induction of the enzymes responsible for initiating their metabolism. While in many cas .... Polycyclic aromatic hydrocarbons (PAHs) are prevalent environmental pollutants which cause a wide range of deleterious health effects. Metabolic activation of PAHs occurs primarily through a set of intracellular oxidising enzymes which are induced by the presence of PAHs. The dioxin receptor is a gene regulatory protein that is pivotal in the metabolic pathway as it links the presence of contaminating PAHs to induction of the enzymes responsible for initiating their metabolism. While in many cases PAHs are oxidised to compounds which are water soluble and excretable (and therefore harmless), some substrates, such as benzo[a]pyrene found in cigarette smoke, can become inadvertently transformed into carcinogens. Other pollutants such as dioxin are resistant to metabolism and are extremely toxic. We have an ongoing interest in deciphering the biochemical pathways which lead to aberrant metabolism, and as such are studying the mechanistic role of the dioxin receptor in this process.
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    Funded Activity

    Signal Transduction Pathways Of EphA Receptor Tyrosine Kinases

    Funder
    National Health and Medical Research Council
    Funding Amount
    $182,249.00
    More information
    Funded Activity

    Defining The Importance Of The AP-1 Transcription Factor In STAT1-independent Functions Of Interferon Gamma.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $307,372.00
    Summary
    Interferons are soluble proteins released by immune cells that play a vital role in immune cell development, their recruitment to sites of injury-infection and detection and removal of pathogens. Interferons act by binding to the surfaces of other cells and altering their behaviour. This project will determine, at a molecular level, how this binding changes cell behaviour. These studies will help to explain why some people are prone to infectious disease.
    More information
    Funded Activity

    Signalling During Red Blood Cell Invasion By Plasmodium Falciparum

    Funder
    National Health and Medical Research Council
    Funding Amount
    $357,414.00
    Summary
    Malaria is one of the world's most devastating infectious diseases and is caused by a parasite called Plasmodium falciparum. AMA1 is a parasite surface protein crucial for blood cell invasion but how it works is not understood. We are investigating if AMA1 plays a role in helping the parasite sense when it has contacted a blood cell and should invade. Discovering how parasites attach to and invade bloods cells is a priority for the development of anti-parasite drugs and vaccines
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    Showing 1-10 of 288 Funded Activites

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