ARDC Research Link Australia Research Link Australia   BETA Research
Link
Australia
  • ARDC Newsletter Subscribe
  • Contact Us
  • Home
  • About
  • Feedback
  • Explore Collaborations
  • Researcher
  • Funded Activity
  • Organisation
  • Researcher
  • Funded Activity
  • Organisation
  • Researcher
  • Funded Activity
  • Organisation

Need help searching? View our Search Guide.

Advanced Search

Current Selection
Research Topic : INSULIN RESISTENCE
Field of Research : Endocrinology
Clear All
Filter by Field of Research
Endocrinology (116)
Filter by Socio-Economic Objective
Search did not return any results.
Filter by Funding Provider
National Health and Medical Research Council (116)
Filter by Status
Closed (116)
Filter by Scheme
NHMRC Project Grants (51)
Project Grants (29)
Postgraduate Scholarships (11)
Early Career Fellowships (9)
NHMRC Postgraduate Scholarships (7)
Career Development Fellowships (4)
NHMRC Development Grants (1)
NHMRC Research Fellowships (1)
Practitioner Fellowships (1)
Program Grants (1)
Research Fellowships (1)
Filter by Country
Australia (5)
Filter by Australian State/Territory
VIC (4)
NSW (1)
SA (1)
  • Researchers (0)
  • Funded Activities (116)
  • Organisations (0)
  • Funded Activity

    Insulin Resistance, Diabetes And Cancer

    Funder
    National Health and Medical Research Council
    Funding Amount
    $150,799.00
    Summary
    The prevalence of type 2 diabetes in increasing worldwide, the International Diabetes Federation predicting 435 million will have diabetes in 2030. The major driver of the diabetes epidemic is obesity. There is strong evidence linking type 2 diabetes and obesity to an increased risk of cancer. However, the exact mechanism promoting cancer development in obese and diabetic individuals is not clear. This project will examine the effects of high insulin levels on cancer development and progression.
    More information
    Funded Activity

    Can Vitamin D Prevent Diabetes By Improving Insulin Sensitivity And Secreation In Overweight Humans?

    Funder
    National Health and Medical Research Council
    Funding Amount
    $715,371.00
    Summary
    Vitamin D is mainly produced by the action of sunlight on skin. Low levels of vitamin D are becoming more common, as we work more indoors and avoid the sun and have been associated with type 2 diabetes. In proposed study, healthy overweight people, with low vitamin D, will be given vitamin D for 16 weeks to optimise blood levels of this vitamin. We will examine the changes in sugar metabolism. This study should provide important evidence on how vitamin D protects against diabetes and will suppor .... Vitamin D is mainly produced by the action of sunlight on skin. Low levels of vitamin D are becoming more common, as we work more indoors and avoid the sun and have been associated with type 2 diabetes. In proposed study, healthy overweight people, with low vitamin D, will be given vitamin D for 16 weeks to optimise blood levels of this vitamin. We will examine the changes in sugar metabolism. This study should provide important evidence on how vitamin D protects against diabetes and will support novel approaches to diabetes prevention.
    Read more Read less
    More information
    Funded Activity

    Can Elevated Insulin Levels Trigger A Loss Of Insulin Response In Cells?

    Funder
    National Health and Medical Research Council
    Funding Amount
    $79,204.00
    More information
    Funded Activity

    Central And Peripheral Actions Of Insulin For The Control Of Muscle Capillary Recruitment

    Funder
    National Health and Medical Research Council
    Funding Amount
    $433,973.00
    Summary
    Type 2 diabetes is on the increase world wide and reflects the ever-increasing incidence of obesity. Whereas the likely cause of type 2 diabetes includes low physical activity and high fat diet, the primary metabolic abnormality is likely to be muscle insulin resistance. The cause of this resistance is controversial, but may stem from microvascular dysfunction where muscle becomes poorly perfused and unresponsive to the action of insulin to recruit capillary flow. In this project we will further .... Type 2 diabetes is on the increase world wide and reflects the ever-increasing incidence of obesity. Whereas the likely cause of type 2 diabetes includes low physical activity and high fat diet, the primary metabolic abnormality is likely to be muscle insulin resistance. The cause of this resistance is controversial, but may stem from microvascular dysfunction where muscle becomes poorly perfused and unresponsive to the action of insulin to recruit capillary flow. In this project we will further extend our seminal discoveries that insulin mediates capillary recruitment under normal circumstances and that in various models of insulin resistance insulin's ability to increase the perfusion of muscle is markedly impaired. We will explore the hypothesis, that insulin controls microvascular perfusion of muscle by a central neural mechanism ending at terminal arterioles on the vasculature and endeavour to identify the details of this control. We will use in-house novel techniques for examining both the role of central control mechanisms involving the brain as well as peripheral mechanisms by local infusion of various agents likely to either enhance or block insulin's microvascular action. A positive outcome will enhance our understanding of insulin action and the insulin resistance that precedes type 2 diabetes. There is also the possible outcome that important clues will be obtained leading to new therapeutic agents that could be used to treat type 2 diabetes.
    Read more Read less
    More information
    Funded Activity

    Hepatic Oxidative Stress, PTPs & STAT Signalling In Obesity

    Funder
    National Health and Medical Research Council
    Funding Amount
    $1,086,547.00
    Summary
    Obesity is increasing at an alarming rate worldwide and is a leading cause of morbidity and mortality. Obesity is causally linked to the development of insulin resistance, a prelude to type 2 diabetes. In this proposal we will define a novel liver centric mechanism by which insulin resistance and oxidative stress may promote the development of morbid obesity, type 2 diabetes and liver disease.
    More information
    Funded Activity

    The Role Of Grb10 In The Regulation Of Muscle Metabolism

    Funder
    National Health and Medical Research Council
    Funding Amount
    $624,960.00
    Summary
    Obesity increases the risk of metabolic diseases such as type 2 diabetes. Muscle is a key tissue for balancing whether energy is used or stored as fat and as we age, muscle mass normally decreases making maintaining a healthy metabolism even more difficult. We have discovered that removing the Grb10 gene from mice produces bigger muscles. This project will investigate the mechanisms of this effect so that strategies can be developed to regulate muscle mass and improve metabolic health
    More information
    Funded Activity

    Effects Of Diet And Exercise On The Mechanisms Of Hyperandrogenisation, Insulin Resistance And LH Pulsatility

    Funder
    National Health and Medical Research Council
    Funding Amount
    $254,910.00
    More information
    Funded Activity

    Intervening In The Natural History Of Type 1 Diabetes: An Integrated Approach

    Funder
    National Health and Medical Research Council
    Funding Amount
    $9,466,000.00
    Summary
    This Program brings together four of Australia’s top type 1 diabetes clinical and lab-based research teams. The program has three intersecting themes. The first theme, pathogenesis, focuses on early life and understanding why type 1 diabetes develops. The second theme, prevention, seeks to identifying new drugs to stop the disease from occurring. The third theme, treatment, aims to improve therapies to replace the cells that are destroyed during the disease process.
    More information
    Funded Activity

    Transforming Mealtime Insulin Dosing Using An Innovative New Bolusing Calculator To Optimise Glycaemic Control In Type 1 Diabetes

    Funder
    National Health and Medical Research Council
    Funding Amount
    $318,768.00
    Summary
    People with type 1 diabetes require life-long insulin injections to maintain their blood glucose levels in the optimal range. Mealtime dose estimations based solely on carbohydrate have limited efficacy as they fail to take fat and protein into account. The aim of this project is to develop an evidence-based insulin bolusing calculator integrated into a smartphone app to enhance blood glucose control and reduce the daily burden of disease and the risk of life-threatening complications.
    More information
    Funded Activity

    Free-Living Closed-Loop Insulin Delivery For Patients With Type 1 Diabetes: A Long-term Multi-centre Randomized Controlled Trial

    Funder
    National Health and Medical Research Council
    Funding Amount
    $1,041,986.00
    Summary
    Closed loop technology for automating the delivery of insulin to patients with Type 1 diabetes has the potential to improve the lives of many with the disease. Though early prototypes are currently under evaluation in small studies, no studies have yet to date applied the technology in a large scale randomised trial. We propose to undertake such a study in order to demonstrate the utility of this technology and facilitate its eventual routine and widespread use in the community in the future.
    More information

    Showing 1-10 of 116 Funded Activites

    • 1
    • 2
    • 3
    • 4
    • 5
    Advanced Search

    Advanced search on the Researcher index.

    Advanced search on the Funded Activity index.

    Advanced search on the Organisation index.

    National Collaborative Research Infrastructure Strategy

    The Australian Research Data Commons is enabled by NCRIS.

    ARDC CONNECT NEWSLETTER

    Subscribe to the ARDC Connect Newsletter to keep up-to-date with the latest digital research news, events, resources, career opportunities and more.

    Subscribe

    Quick Links

    • Home
    • About Research Link Australia
    • Product Roadmap
    • Documentation
    • Disclaimer
    • Contact ARDC

    We acknowledge and celebrate the First Australians on whose traditional lands we live and work, and we pay our respects to Elders past, present and emerging.

    Copyright © ARDC. ACN 633 798 857 Terms and Conditions Privacy Policy Accessibility Statement
    Top
    Quick Feedback