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Research Topic : CYTOKINE REGULATION
Field of Research : Cancer Cell Biology
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Cancer Cell Biology (24)
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  • Researchers (4)
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  • Funded Activity

    Does CD123 Provide A Biological Advantage To Leukaemia Stem Cells?

    Funder
    National Health and Medical Research Council
    Funding Amount
    $647,637.00
    Summary
    Leukaemia is a devastating form of blood cancer affecting both young and old. We need to understand the diseased stem cell to eradicate this disease. Current therapy is poorly tolerated and the majority of patients ultimately die at relapse. We intend to investigate how we can make the cells more susceptible to therapy by understanding their biology.
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    Funded Activity

    Regulation Of Innate Immunity And Tumour Progression By Activating Transcription Factor 3

    Funder
    National Health and Medical Research Council
    Funding Amount
    $473,469.00
    Summary
    Toll-like receptors (TLRs) play an essential role in innate immune responses and are involved in initiating tumourigenesis via inflammatory pathways. We have shown that the transcription factor ATF3 is a negative regulator of TLR signalling. We will study how modulation of the activity of ATF3 affects the inflammatory response and tumour progression. This will provide a molecular basis on which to design therapeutic reagents for the treatment of cancer.
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    Funded Activity

    Investigating The Role Of HoxB8 In The Development Of Acute Myeloid Leukaemia (AML).

    Funder
    National Health and Medical Research Council
    Funding Amount
    $36,533.00
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    Funded Activity

    Therapeutically Exploiting Non-oncogene Addiction And Defining Genetic Interactions For Disease Progression In A Preclinical Model Of Inflammation-dependent Gastric Tumourigenesis

    Funder
    National Health and Medical Research Council
    Funding Amount
    $624,960.00
    Summary
    Cancers of the stomach are often associated with chronic inflammation and represent a major health burden with little treatment options available. We propose to test whether drugs undergoing clinical testing for other diseases may have beneficial effects in a preclinical model of gastric cancer, and establish the genetic interaction required for gastric cancer progression. The study outcomes may highlight novel therapeutic opportunities for the clinic.
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    Funded Activity

    Research Fellowship

    Funder
    National Health and Medical Research Council
    Funding Amount
    $675,736.00
    Summary
    I am a cancer biologist determining the mechanisms controlling growth and proliferation of cancer cells and use transgenic models of malignancy and genetic approaches to identify new therapies for targeting growth control in the treatment of cancer.
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    Funded Activity

    Transcriptional Regulation Of The Expression Of Mcl - 1 In Melanoma Cell Under ER Stress

    Funder
    National Health and Medical Research Council
    Funding Amount
    $152,194.00
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    Funded Activity

    Novel Targeting Of Therapy-resistant Prostate Cancer Cells.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $596,978.00
    Summary
    Prostate cancer is treated by removing male hormones (androgens). Although the bulk of the tumour regresses, some cells remain and the cancer often grows back in an aggressive form. We will study new ways to eliminate therapy resistant cancer cells and thereby provide more lasting treatments for prostate cancer. Ultimately, we hope to inform the design of ground-breaking clinical trials that could re-shape the treatment paradigm of advanced prostate cancer.
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    Funded Activity

    To Determine Whether Myc-driven Transformation In Haematopoietic Cell Lineages Is Dependent On High-levels Of Myc Protein Expression.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $371,896.00
    Summary
    Myc is an essential cellular protein but is also a common drive of cancer in multiple tissues. In blood cancers Myc is frequently overexpressed. In contrast, Myc is rarely overexpressed in early stage solid cancers, although often elevated levels at later stages. We will employ unique models of cancer in which Myc can be activated at different set levels at different times during blood cell development to address what the specific contributions of different levels of Myc are in the evolution of .... Myc is an essential cellular protein but is also a common drive of cancer in multiple tissues. In blood cancers Myc is frequently overexpressed. In contrast, Myc is rarely overexpressed in early stage solid cancers, although often elevated levels at later stages. We will employ unique models of cancer in which Myc can be activated at different set levels at different times during blood cell development to address what the specific contributions of different levels of Myc are in the evolution of blood cancers.
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    Funded Activity

    Therapeutic Targeting Of Ribosome Biogenesis In Cancer And Ribosomopathies

    Funder
    National Health and Medical Research Council
    Funding Amount
    $763,845.00
    Summary
    My fellowship application will build on my international leadership in understanding growth control in human disease. My vision is to uncover the molecular mechanisms governing the loss of normal control of the synthesis of the molecular machines, termed ribosomes, that are responsible for synthesising all cell proteins. I will translate these findings into new paradigms to treat patients suffering from diseases such as cancer and ribosomopathies, that are associated with ribosome dysfunction.
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    Funded Activity

    Restoration Of P53 Activity In Tumours: A New Approach Involving The P53 Coactivator ANKRD11.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $465,990.00
    Summary
    p53 is an important protein that functions as the body�s defence mechanism against cancer. Mutation of p53 is observed in over half of all tumours. Not only do these cancer mutations abolish the ability of p53 to protect against cancer, but it also endows the tumours with an ability to spread throughout the body, or metastasize. In this research project, we will identify and develop targets that will not only prevent the spread of new tumours, but it will also re-activate the anti-cancer functio .... p53 is an important protein that functions as the body�s defence mechanism against cancer. Mutation of p53 is observed in over half of all tumours. Not only do these cancer mutations abolish the ability of p53 to protect against cancer, but it also endows the tumours with an ability to spread throughout the body, or metastasize. In this research project, we will identify and develop targets that will not only prevent the spread of new tumours, but it will also re-activate the anti-cancer function in mutant p53 leading to tumour regression.
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    Showing 1-10 of 24 Funded Activites

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