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Research Topic : COPD pathophysiology
Scheme : Project Grants
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  • Funded Activity

    Characterising A Newly Identified Mechanism Causing Elevation Of Intracranial Pressure After Acute Neurological Injury

    Funder
    National Health and Medical Research Council
    Funding Amount
    $510,905.00
    Summary
    Our group discovered that increased pressure on the brain (intracranial pressure – ICP) may be more common and important than has been recognised following stroke, and potentially other brain disorders. We also identified a simple potential therapy, short-duration body cooling, which completely prevents the pressure rise. In this project we will characterise the ICP rise, identify its molecular trigger and determine the best method of body cooling tor use in clinical trials in stroke patients.
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    Funded Activity

    Clarifying The Pathogenic Role Of Arousal-hyperventilation In Obstructive And Central Sleep Apnoea: Testing Fundamental Pathophysiological Mechanisms And A Strategic New Treatment

    Funder
    National Health and Medical Research Council
    Funding Amount
    $414,717.00
    Summary
    This project is designed to understand the mechanisms underpinning much more stable breathing during deep sleep in obstructive sleep apnoea (OSA). A newly developed analytical technique will be used to examine breathing effort changes across sleep, and interactions with respiratory-induced awakenings in OSA patients. In addition, a new treatment designed to stabilise breathing will be tested and refined towards a new treatment option for OSA and for central sleep apnoea.
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    Funded Activity

    Elucidating The Role And Potential For Therapeutic Targeting Of TLR7 In Emphysema And COPD

    Funder
    National Health and Medical Research Council
    Funding Amount
    $925,780.00
    Summary
    Emphysema is a major cause of illness and death and there are no effective treatments. It is caused by smoking that damages the airways and air sacs but how this occurs is not well understood. We have found that a new factor, called Toll-like receptor 7, is involved in emphysema. In this study we will now characterise its roles in this disease, work out how it induces emphysema and test new inhibitors (antibodies) that we have developed as treatments.
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    Funded Activity

    Studies On The Effects Of Endogenous And Exogenous Opioids In Modulating Exercise-induced Dyspnoea In People With Moderate / Severe Chronic Obstructive Pulmonary Disease (COPD). Two Double-blind Randomised, Placebo Controlled Trials.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $197,086.00
    Summary
    There is evidence that morphine-like chemicals made by the body itself help reduce breathlessness. It is not clear whether this response is because of the effects of these morphine-like chemicals on the brain, in the lung or both. This study will help understand where these chemicals act to reduce breathlessness in people with emphysema during exercise. Subsequently, it will also help to understand whether small, regular doses of morphine safely further reduce breathlessness during exercise.
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    Funded Activity

    Enhancing Innate Immune Responses To Influenza In Chronic Obstructive Pulmonary Disease

    Funder
    National Health and Medical Research Council
    Funding Amount
    $522,323.00
    Summary
    Infection with influenza is a serious health problem for all, but particularly those with chronic lung diseases such as chronic obstructive pulmonary disease (COPD). Current treatments are limited. We have previously shown that human COPD airway cells are more susceptible to infection with influenza and the virus is able to effectively block the early immune response. We propose to define the mechanism that underlie this and design novel inhibitors to enhance the early immune response of these c .... Infection with influenza is a serious health problem for all, but particularly those with chronic lung diseases such as chronic obstructive pulmonary disease (COPD). Current treatments are limited. We have previously shown that human COPD airway cells are more susceptible to infection with influenza and the virus is able to effectively block the early immune response. We propose to define the mechanism that underlie this and design novel inhibitors to enhance the early immune response of these cells and reduce the effects of infection.
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    Funded Activity

    Understanding The Role Of Th22 Cells In Regulating Respiratory Immune Responses In Health And Disease.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $870,476.00
    Summary
    T cells that produce the cytokine IL-22 (Th22 cells) are found in infectious and inflammatory lung disease. However, the role of Th22 cells in promoting or preventing disease remains largely unknown. We have discovered how to grow Th22 cells and have generated a unique strain of IL-22 reporter mice, which will allow us to identify their role in infectious and inflammatory diseases. Our investigations will provide new insights into therapeutic approaches for these diseases of the lung.
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    Funded Activity

    Defining The Roles And Targeting Interferon-epsilon As A New Therapy For Influenza In Asthma And COPD

    Funder
    National Health and Medical Research Council
    Funding Amount
    $905,904.00
    Summary
    Influenza is a major cause of illness and death, especially in people with asthma and emphysema. There are issues with vaccines and current treatments are poorly effective. Effective treatments are urgently required. We have found a new immune factor, interferon-epsilon that is induced and used by influenza viruses to cause infection. We aim to understand how this occurs and to test new treatments for influenza that suppress interferon-epsilon, in healthy and susceptible individuals.
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    Funded Activity

    Exploiting Increased Autophagy In Bronchial Epithelial Cells: A New Therapeutic Approach For Chronic Obstructive Pulmonary Disease (COPD)

    Funder
    National Health and Medical Research Council
    Funding Amount
    $724,161.00
    Summary
    COPD is incurable, a leading cause of death, and new therapies are urgently needed. Autophagy is a cell response to cell stress conditions, however increased autophagy is harmful. We will investigate the association of increased autophagy with COPD and smoking and evaluate therapies that can reduce autophagy, including zinc-related drugs and novel antibiotics that have been modified to lose their anti-bacterial activity.
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    Funded Activity

    Characterising The Pro-inflammatory Role Of IL-36γ/IL-36R In Pathogen-induced Exacerbations Of Asthma And COPD

    Funder
    National Health and Medical Research Council
    Funding Amount
    $738,768.00
    Summary
    Lung infections cause exacerbations in patients with asthma and COPD and these are difficult to treat and can be life-threatening. We have discovered that the cytokine pathway, IL-36?/IL-36R, is linked to the host response to bacterial and viral infections that induce exacerbations in these patients. Our studies will provide new insight into the role IL-36?/IL-36R in infection, disease exacerbation and determine the therapeutic benefit of blocking this pathway.
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    Funded Activity

    Testing The Lung Microbiome To Predict Risk Of Frequent Exacerbations In COPD

    Funder
    National Health and Medical Research Council
    Funding Amount
    $666,052.00
    Summary
    Chronic obstructive pulmonary disease (COPD) is a disease of global importance. Exacerbations, mostly due to respiratory infection, are complications that lead to significant illness. This study will characterise the communities of microbes in the lung, and use this information to predict frequency of exacerbations of COPD measured over 12 months.
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    Showing 1-10 of 51 Funded Activites

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