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Research Topic : APOBEC3G restriction factor
Australian State/Territory : VIC
Scheme : Project Grants
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  • Funded Activity

    Reducing Morbidities In Preterm Growth Restricted Neonates.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $687,214.00
    Summary
    Intrauterine growth restriction (IUGR) is a serious complication of pregnancy and occurs when fetal growth is abnormal, resulting in a fetus that is smaller than it should be for its given gestational age. IUGR babies are at much greater risk of many short and long-term adverse outcomes. This study investigates the role that adverse cardiovascular development plays in the progression of lung, heart and brain disease in preterm IUGR newborns.
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    Funded Activity

    Measuring Hypoxia Induced MRNA In Maternal Blood To Monitor Wellbeing Of Growth-restricted Fetuses

    Funder
    National Health and Medical Research Council
    Funding Amount
    $421,358.00
    Summary
    Severely growth restricted fetuses are at peril of stillbirth from low oxygenation. While ultrasound monitoring improves outcomes, babies are still lost. Better ways to monitor the health the unborn baby are needed. We have recently discovered fetuses’ starved of oxygen leak RNA into mother's blood. Thus, measuring RNA molecules in blood could be used to assess fetal health. We will examine whether measuring mRNA in maternal blood could be used to monitor wellbeing of growth-restricted fetuses.
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    Funded Activity

    Improving The Prediction And Detection Of Contributors To Term Stillbirth

    Funder
    National Health and Medical Research Council
    Funding Amount
    $570,358.00
    Summary
    Stillbirths are a global human tragedy, with 1 in 130 of all pregnancies in Australia ending in stillbirth. We propose to use ultrasound and blood markers to improve the detection of babies who are not growing well, a leading risk factor for stillbirth. Sleep position has also been associated with stillbirth, so we will study fetal heart rate responses during an overnight sleep study to see if breathing events overnight may be an important contributor to stillbirth in growth restricted fetuses.
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    Funded Activity

    Roles Of The EMT Transcription Factors In Epigenetic Remodelling And Myeloid Cell Transformation.

    Funder
    National Health and Medical Research Council
    Funding Amount
    $809,520.00
    Summary
    This project is based upon our novel discoveries that identified ZEB2 and SNAI1 as novel genes involved in the development of aggressive forms of blood cancer. During the course of this proposal we will find new drug targets and new drug treatment options using existing drugs that will specifically target cancer initiating cells in order to kill aggressive forms of blood cancers that are currently refractory to treatment.
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    Funded Activity

    Transcriptional Effectors Of Oncogenic ERK Signaling In Colorectal Cancer

    Funder
    National Health and Medical Research Council
    Funding Amount
    $820,776.00
    Summary
    This project aims to unravel how one of the most frequently deregulated molecular pathways in colorectal cancer controls the expression of genes required for these tumours to grow and spread. We expect this work to uncover novel therapeutic targets to effectively inactivate this pathway and biomarkers to select patients most likely to benefit from existing therapies.
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    Funded Activity

    EPIGENETIC REPROGRAMMING OF MALIGNANT BREAST CANCER

    Funder
    National Health and Medical Research Council
    Funding Amount
    $863,268.00
    Summary
    Poorly differentiated breast cancers are aggressive tumors, frequently resistant to chemotherapy and associated with high morbidity. Herein we propose the engineering of more selective therapeutic agents able to target the genes involved in cancer initiation and resistance to treatment. We aim to correct and reprogram the cancer cell genome in state that is similar to normal, not tumorigenic cells. This work will generate novel forms of treatment for cancers that are presently not curable.
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    Funded Activity

    Molecular Basis For Stress-induced Gene Regulation—a Model System To Understand Transcriptional Deregulation In Cancer And Neurological Disease

    Funder
    National Health and Medical Research Council
    Funding Amount
    $384,076.00
    Summary
    Deregulated gene transcription plays a critical role in cancer formation. It is therefore important to understand the molecular basis of gene transcription and how tumour cells hijack the process. In this Project, we will study the molecular basis of stress-inducible gene expression. This is particularly important for understanding the molecular basis of cancer as stress-inducible genes are activated by transcription factors implicated in breast, colon, lung, and prostate cancers.
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    Funded Activity

    Novel Strategies To Promote Myelin Repair In The Brain

    Funder
    National Health and Medical Research Council
    Funding Amount
    $597,865.00
    Summary
    Demyelinating diseases of the central nervous system such as multiple sclerosis have a lifelong impact and devastating impact on quality of life. We have identified that a growth factor, brain derived neurotrophic factor (BDNF), plays an important role in promoting myelination during development. We will investigate the potential of translating these findings into effective clinical treatment, by characterising the efficacy of BDNF in promoting CNS remyelination after a demyelinating insult.
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    Funded Activity

    New Drug Combinations To Enhance Elimination Of Hepatitis B Infection

    Funder
    National Health and Medical Research Council
    Funding Amount
    $888,304.00
    Summary
    We have developed a therapy that kills hepatitis B virus infected cells and promotes elimination of infection. We are now testing novel drugs that can be used to maximise the efficacy of our new treatment to promote better outcomes that may be translated to other infections.
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    Funded Activity

    Regulation Of TNF Expression In Inflammation And Cancer

    Funder
    National Health and Medical Research Council
    Funding Amount
    $728,447.00
    Summary
    By studying a spontaneous mutation in mice, we have found an error in the TNF gene (a major factor in many inflammatory diseases) that causes severe arthritis, heart valve disease and gut inflammation. We have also identified new regulators of TNF expression, which might be useful therapeutic targets to limit inflammation. We intend to study the role of these regulators in controlling the expression of TNF, and the link between chronic inflammation and the development of cancer.
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