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Research Topic : ALTERNATIVE SPLICING
Scheme : NHMRC Project Grants
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  • Funded Activity

    Effects Of Telomerase Catalytic Subunit On Human Cells

    Funder
    National Health and Medical Research Council
    Funding Amount
    $281,347.00
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    Funded Activity

    Therapeutic Induction Of Dytrophin-positive Revertant Fibres In The Mdx Mouse

    Funder
    National Health and Medical Research Council
    Funding Amount
    $454,825.00
    Summary
    Revertant fibres are low-abundance, dystrophin-positive fibres found in muscle of DMD patients and animal models. These fibres appear to have a selective advantage over dystrophin negative fibres, as they accumulate with age. Characterisation of dystrophin mRNA has identified in-frame transcripts missing multiple exons, which either exclude a nonsense mutation or restore the reading frame around a deletion. We have designed antisense oligonucleotides (AOs) to bind regions flanking the exon conta .... Revertant fibres are low-abundance, dystrophin-positive fibres found in muscle of DMD patients and animal models. These fibres appear to have a selective advantage over dystrophin negative fibres, as they accumulate with age. Characterisation of dystrophin mRNA has identified in-frame transcripts missing multiple exons, which either exclude a nonsense mutation or restore the reading frame around a deletion. We have designed antisense oligonucleotides (AOs) to bind regions flanking the exon containing the dystrophin mutation in the mdx mouse. The AOs interfere with processing of the pre-mRNA to exclude the mutation and allow a slightly shortened dystrophin to be synthesised. The use of AOs to modify RNA processing allows the gene to function under the control of natural regulatory elements. We have shown that AOs can induce dystrophin expression and improve strength in dystrophic (mdx) mouse hindlimb muscles. We aim to improve upon these results by using AOs to block splice sites flanking consecutive exons, in order to induce dystrophin which mimics that of revertant fibres. As most revertant transcripts are missing multiple exons, we believe that the functional capacity of AO-induced dystrophin can be improved upon by removing multiple exons. An mdx mouse skeletal muscle cell line is used for evaluation AOs. However, in order to determine the efficacy of the induced dystrophin in cardiac and skeletal muscle, experiments must be performed on mice. Previous work, in vitro and in muscles of mdx mice have validated this approach. Combinations of AOs which show promise will be delivered by a) intravascular injection b) intraperitoneal injection in mdx mice. The efficacy of the treatment will be assessed by both continual and end point analysis, which includes physiological, clinical, molecular and histological testing. Particular attention will be directed to the well-being of the mice and any adverse side effects which may occur.
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    Funded Activity

    Function Of Collagen VI In Tissue Development

    Funder
    National Health and Medical Research Council
    Funding Amount
    $184,524.00
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    Funded Activity

    Haemopoetic Molecular And Cellular Analysis Of Human Genes At 11P15.5

    Funder
    National Health and Medical Research Council
    Funding Amount
    $172,400.00
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    Funded Activity

    The Role Of Growth Factor Depots In Repair Of Injury To Blood Vessels

    Funder
    National Health and Medical Research Council
    Funding Amount
    $89,257.00
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    Funded Activity

    Studies On The Origin Of Dystrophin Within A Dystrophin Negative Muscle Tissue

    Funder
    National Health and Medical Research Council
    Funding Amount
    $84,963.00
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    Funded Activity

    Structure And Function Of The Alternative Splicing Factor ZNF265

    Funder
    National Health and Medical Research Council
    Funding Amount
    $509,017.00
    Summary
    Now that the human genome has been sequenced, we can see that a human being is defined bye approximately 30000 genes. One of the biggest surprises to come from this work was that the number of genes was significantly smaller than many predicted. Similar surprise was registered at the discovery that the genome of the fruit fly actually contained fewer genes than that of the model worm, Caenorhabditis elegans. Part of the explanation for these apparent discrepencies lies in the phenomenon known as .... Now that the human genome has been sequenced, we can see that a human being is defined bye approximately 30000 genes. One of the biggest surprises to come from this work was that the number of genes was significantly smaller than many predicted. Similar surprise was registered at the discovery that the genome of the fruit fly actually contained fewer genes than that of the model worm, Caenorhabditis elegans. Part of the explanation for these apparent discrepencies lies in the phenomenon known as gene splicing, whereby one gene can actually give rise to many different isoforms of the same protein. These different isoforms can have different structures and-or functions, and dramatically increase the complexity that it is possible for an organism to achieve with a given number of genes. The process of splicing is very intricate, requiring precise control to allow an organism to develop normally. Many human genetic diseases are known to arise from problems with splicing. However, our understanding of the mechanisms of splicing is rather incomplete. This proposal aims to improve our understanding of the splicing process through a range of biophysical and molecular biological approaches. This information should prove useful in understanding human development and disease.
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    Funded Activity

    Alternate Splicing Of Tryptase Genes Regulates Their Specificity

    Funder
    National Health and Medical Research Council
    Funding Amount
    $294,250.00
    Summary
    Tryptases are enzymes implicated in inflammatory disorders including arthritis, inflammatory bowel disease (IBD) and asthma. Specific tryptase inhibitors are effective in treating these diseases. We have discovered that each human tryptase gene is processed into two different protein products via a mechanism called alternate splicing. We will investigate the structure and function of these.
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    Funded Activity

    Pathophysiology And Alternative Preventative Strategy For Breast Cancer Chemotherapy-induced Bone Loss

    Funder
    National Health and Medical Research Council
    Funding Amount
    $540,356.00
    Summary
    Combination cytotoxic chemotherapy is the current optimal approach for treating breast cancer in premenopausal women. However, long-term skeletal defects (osteoporosis and fractures) caused by the chemotherapy have become an increasingly serious problem due to its intensified use and improved patient survival rate. This project seeks to elucidate the mechanisms for chemotherapy-induced bone defects and to initiate development of a preventative treatment using natural bioactive micronutrients.
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    Funded Activity

    The Efficacy And Safety Of Acupuncture For Seasonal Allergic Rhinitis: A Prospective, Randomised Controlled Trial

    Funder
    National Health and Medical Research Council
    Funding Amount
    $175,732.00
    Summary
    Seasonal allergic rhinitis, also known as hay fever, is a common condition with high prevalence in Australia, being most pronounced during spring and early summer when pollen counts are highest. There is no proven cure for hay fever. Many hay fever sufferers seek alternative therapies such as acupuncture. We propose to undertake a rigorous study, using well-established clinical trial methodology, to evaluate the efficacy and safety of acupuncture in the treatment of hay fever.
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    Showing 1-10 of 20 Funded Activites

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